MétaCan
Menu
← Back to cohort
Record W2047942417 · doi:10.1158/1538-7445.am10-5049

Abstract 5049: Functional significance of <i>L1CAM</i> in non-small cell lung cancer

2010· article· en· W2047942417 on OpenAlexaffabout
Josephine Hai, Nikolina Radulovich, Chang‐Qi Zhu, Sandy D. Der, Igor Jurišica, Ming‐Sound Tsao

Bibliographic record

VenueCancer Research · 2010
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicCancer, Hypoxia, and Metabolism
Canadian institutionsOntario Institute for Cancer Research
Fundersnot available
KeywordsLung cancerMedicineCancer researchOncologyCarcinogenesisSmall hairpin RNAInternal medicineGene knockdownCancerMatrigelMetastasisPathologyCell cultureBiology

Abstract

fetched live from OpenAlex

Abstract Background: Non-small cell lung cancer (NSCLC) accounts for 80% of all lung cancers. Early stage NSCLC patients are treated by complete surgical resection, but the 5-year survival rate remains relatively poor at 50%. Deaths are mainly due to metastatic recurrence. Post-surgery (adjuvant) chemotherapy may significantly improve survival, but improved selection methods for patients who are most likely to benefit from the adjuvant treatment are required. Our laboratory has recently identified a 15-gene expression signature capable of classifying patients based on risk of death and at the same time predicting benefits of adjuvant chemotherapy in high-risk patients (J Clin Oncol 26: 2008, May 20 suppl; abstr 7510). We hypothesize that genes whose expression are de-regulated significantly in patients with a poor prognosis may play important roles in tumorigenesis and metastasis. Our goal is to functionally characterize the roles of these prognostic genes in the growth and metastatic potential of NSCLC. One of these signature genes is L1CAM, a cell adhesion molecule. Although extensively characterized in the nervous system, the role of L1CAM in NSCLC has not been defined. Methods: We stably modified the expression of L1CAM at both the RNA and protein levels in NSCLC cell lines using the lentiviral cDNA/shRNA system. In vitro matrigel assays were used to assess invasion and cell migration by wound-healing assays. Results: Downregulation of L1CAM expression resulted in decreased invasiveness and motility of metastatic NSCLC cell lines. In contrast, loss of L1CAM did not alter the growth rate on plastic. Studies on anchorage-independent growth and in vivo tumorigenicity and metastatic potential are ongoing and the results will be presented. Conclusion: Considering the importance of migratory phenotype in the first steps of metastasis, the results suggest that L1CAM contributes to malignancy in NSCLC and may be a novel therapeutic gene for this disease. (Supported by Canadian Cancer Society Research Institute grant #020527) Citation Format: {Authors}. {Abstract title} [abstract]. In: Proceedings of the 101st Annual Meeting of the American Association for Cancer Research; 2010 Apr 17-21; Washington, DC. Philadelphia (PA): AACR; Cancer Res 2010;70(8 Suppl):Abstract nr 5049.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.011

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0030.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.028
GPT teacher head0.335
Teacher spread0.307 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2010
Admission routes2
Has abstractyes

Explore more

Same venueCancer Research→Same topicCancer, Hypoxia, and Metabolism→French-language works237,207→