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Record W2062511407 · doi:10.1371/journal.pgen.1002245

Predisposition to Cancer Caused by Genetic and Functional Defects of Mammalian Atad5

2011· article· en· W2062511407 on OpenAlexafffund
Daphne W. Bell, Nilabja Sikdar, Kyoo‐young Lee, Jessica C. Price, Raghunath Chatterjee, Hee‐Dong Park, Jennifer T. Fox, Masamichi Ishiai, Meghan L. Rudd, Lana M. Pollock, Sarah Fogoros, Hassan Mohamed, Christin L. Hanigan, Suiyuan Zhang, Pedro Cruz, Gabriel Renaud, Nancy F. Hansen, Praveen F. Cherukuri, Bhavesh Borate, Kirk J. McManus, Jan Stoepel, Payal Sipahimalani, Andrew K. Godwin, Dennis C. Sgroi, Maria J. Merino, Gene Elliot, Abdel G. Elkahloun, Charles Vinson, Minoru Takata, James C. Mullikin, Tyra G. Wolfsberg, Philip Hieter, Dae‐Sik Lim, Kyungjae Myung

Bibliographic record

VenuePLoS Genetics · 2011
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicDNA Repair Mechanisms
Canadian institutionsCanada's Michael Smith Genome Sciences CentreUniversity of British Columbia
FundersNational Cancer InstituteNational Institutes of HealthNational Human Genome Research InstituteMassachusetts General HospitalOvarian Cancer Research FundCanadian Institutes of Health ResearchAvon Foundation for Women
KeywordsGenome instabilityBiologyCarcinogenesisDNA damageProliferating cell nuclear antigenCancer researchMutationDNA repairGeneticsSomatic cellDNA mismatch repairNonsense mutationGermline mutationMolecular biologyCancerGeneDNAMissense mutation

Abstract

fetched live from OpenAlex

ATAD5, the human ortholog of yeast Elg1, plays a role in PCNA deubiquitination. Since PCNA modification is important to regulate DNA damage bypass, ATAD5 may be important for suppression of genomic instability in mammals in vivo. To test this hypothesis, we generated heterozygous (Atad5(+/m)) mice that were haploinsuffficient for Atad5. Atad5(+/m) mice displayed high levels of genomic instability in vivo, and Atad5(+/m) mouse embryonic fibroblasts (MEFs) exhibited molecular defects in PCNA deubiquitination in response to DNA damage, as well as DNA damage hypersensitivity and high levels of genomic instability, apoptosis, and aneuploidy. Importantly, 90% of haploinsufficient Atad5(+/m) mice developed tumors, including sarcomas, carcinomas, and adenocarcinomas, between 11 and 20 months of age. High levels of genomic alterations were evident in tumors that arose in the Atad5(+/m) mice. Consistent with a role for Atad5 in suppressing tumorigenesis, we also identified somatic mutations of ATAD5 in 4.6% of sporadic human endometrial tumors, including two nonsense mutations that resulted in loss of proper ATAD5 function. Taken together, our findings indicate that loss-of-function mutations in mammalian Atad5 are sufficient to cause genomic instability and tumorigenesis.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.011
Threshold uncertainty score0.550

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.018
GPT teacher head0.218
Teacher spread0.199 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations99
Published2011
Admission routes2
Has abstractyes

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