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Record W2068276080 · doi:10.1158/1538-7445.am2011-1404

Abstract 1404: L1CAM in non-small-cell lung carcinoma

2011· article· en· W2068276080 on OpenAlexaffabout
Josephine Hai, Chang‐Qi Zhu, Sandy Der, Igor Jurišica, Ming‐Sound Tsao

Bibliographic record

VenueCancer Research · 2011
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicRNA modifications and cancer
Canadian institutionsOntario Institute for Cancer Research
Fundersnot available
KeywordsMedicineVinorelbineCancer researchCisplatinSmall hairpin RNAL1Lung cancerGene knockdownMatrigelCell cultureOncologyChemotherapyPathologyInternal medicineBiologyAngiogenesis

Abstract

fetched live from OpenAlex

Abstract Background: Non-small-cell lung carcinoma (NSCLC) is the predominant type of lung cancer, accounting for 85% of all malignant lung tumors. Early stage (I-II) NSCLC is primarily treated by surgical resection with curative intent, but at least 50% of these patients will experience tumor recurrence and die from it. Recent clinical trials have demonstrated the survival benefit of post-surgery adjuvant chemotherapy in stage II and III patients, but its benefit in stage I remains controversial. Our group has previously reported a 15-gene mRNA expression profile that is predictive of patient prognosis and predictive of benefit from adjuvant chemotherapy by cisplatin and vinorelbine. We hypothesize that these genes may play important roles in malignant progression. One of these prognostic genes is L1CAM, a transmembrane protein. The role of L1CAM in the biology of NSCLC cells has not been clearly defined. Methods: The expression of L1CAM was modified at both the RNA and protein levels in H460, H1264, H125, and H460SM cell lines using the lentiviral cDNA/shRNA system. Matrigel assay was used to assess invasion and cell migration by time-lapse wound-healing assay. Tumorigenicity was assessed by injection of modified cell lines into subcutaneous flank of immunodeficient mice. Results: In vitro studies show that L1CAM expression promotes invasiveness and motility of NSCLC cell lines. In vivo studies demonstrate that knock-down of L1CAM expression reduces tumor growth in immunodeficient mice. Mechanistic studies on L1CAM function are ongoing and the results will be presented. Conclusion: Considering the importance of migratory phenotype during metastasis, the results suggest that L1CAM contribute to malignant progression in NSCLC and may be a novel therapeutic target to treat this disease. (Supported by Canadian Cancer Society Research Institute grant #020527) Citation Format: {Authors}. {Abstract title} [abstract]. In: Proceedings of the 102nd Annual Meeting of the American Association for Cancer Research; 2011 Apr 2-6; Orlando, FL. Philadelphia (PA): AACR; Cancer Res 2011;71(8 Suppl):Abstract nr 1404. doi:10.1158/1538-7445.AM2011-1404

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Not applicable · Consensus signal: none
GenreCandidate signal: Other · Consensus signal: none
Teacher disagreement score0.003
Threshold uncertainty score0.009

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0030.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.080
GPT teacher head0.359
Teacher spread0.280 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designNot applicable
Domainnot available
GenreOther

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2011
Admission routes2
Has abstractyes

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