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Record W2075872996 · doi:10.1158/1538-7445.am2011-1096

Abstract 1096: Prostate tumor growth in pbARR2-Cre PTEN knockout mice is delayed in GH/IGF-I deficient mice

2011· article· en· W2075872996 on OpenAlexaff
Naokazu Ibuki, Kiyoshi Takahara, Howard Tearle, Martin Gleave, Christopher J. Ong, Michaël Pollak, Michael Cox

Bibliographic record

VenueCancer Research · 2011
Typearticle
Languageen
FieldMedicine
TopicProstate Cancer Treatment and Research
Canadian institutionsMcGill University
Fundersnot available
KeywordsPTENProstate cancerBiologyEndocrinologyProstateInternal medicineCancer researchCancerMedicinePI3K/AKT/mTOR pathwaySignal transductionCell biology

Abstract

fetched live from OpenAlex

Abstract Growth hormone/insulin-like growth factor I (GH/IGF-I) axis has been related to risk of prostate cancer (PCa). We have previously reported that androgen-responsive, castration-resistant (CR), and androgen-independent (AI) PCa xenografts develop more slowly in little murine hosts. The little (lit/lit) phenotype is due to a D60G missense mutation in the GH-releasing hormone receptor (GHRHR) resulting in loss of pituitary GHRHR function and suppression of GH and IGF-I expression. PTEN is a tumor suppressor gene that is frequently mutated in a variety of spontaneous cancers, including PCa. The prostate-conditional PTEN-null mouse represents the first animal model in which deletion of a single endogenous gene leads to invasive PCa. To investigate the role of GH/IGF-I axis on in vivo prostate carcinogenesis and neoplastic progression, we crossed pbARR2-Cre, PTEN(fl/fl) mice (PTEN-/-) with IGF-I deficient lit mice, and produced lit/lit and lit/+ PTEN-/- mice. To complement the in vivo experiments, in vitro growth and growth factor signaling of murine PTEN-/- cells derived from the prostate (MPPK) or lymph node (MLPK) using serum from lit/lit or lit/+ mice was examined. RESULTS: Body weight and serum GH, IGF-I levels of lit/lit PTEN-/- mice were significantly reduced as compared with lit/+ PTEN-/- mice. At 15 weeks of age, prostate cancer progression of lit/lit PTEN-/- mice was partially delayed as compared with lit/+ PTEN-/- mice. MPPK and MLPK cells growth in vitro with serum from lit/lit mice showed decreased proliferation as compared with serum from lit/+ mice. Suppressed growth in lit/lit serum could be restored by addition of IGF-I, and to a lesser extent, GH. Addition of GH or IGF-I to lit/lit serum increased only activation of AKT while no change in ERK1/2 activation was detected in MPPK cells. Our in vivo and in vitro results suggest that prostate carcinogenesis may be influenced by germ line variation of genes encoding signaling molecules in the GH/IGF-I axis and support the continued development of clinical trials of novel hormonal treatment strategies that target the GH/IGF-I axis for PCa patients. Citation Format: {Authors}. {Abstract title} [abstract]. In: Proceedings of the 102nd Annual Meeting of the American Association for Cancer Research; 2011 Apr 2-6; Orlando, FL. Philadelphia (PA): AACR; Cancer Res 2011;71(8 Suppl):Abstract nr 1096. doi:10.1158/1538-7445.AM2011-1096

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.009
Threshold uncertainty score0.029

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.001
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0010.000
Science and technology studies0.0000.001
Scholarly communication0.0010.001
Open science0.0010.000
Research integrity0.0010.003
Insufficient payload (model declined to judge)0.0090.003

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.106
GPT teacher head0.398
Teacher spread0.293 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2011
Admission routes1
Has abstractyes

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