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Record W2093673501 · doi:10.1113/jphysiol.2013.257626

Short‐term exercise training augments α<sub>2</sub>‐adrenoreceptor‐mediated sympathetic vasoconstriction in resting and contracting skeletal muscle

2013· article· en· W2093673501 on OpenAlexaff
Nicholas Jendzjowsky, Darren S. DeLorey

Bibliographic record

VenueThe Journal of Physiology · 2013
Typearticle
Languageen
FieldMedicine
TopicHeart Rate Variability and Autonomic Control
Canadian institutionsUniversity of Alberta
Fundersnot available
KeywordsVasoconstrictionSkeletal muscleTraining (meteorology)Internal medicineSympathetic nervous systemTerm (time)MedicineCardiologyChemistryEndocrinologyBlood pressure

Abstract

fetched live from OpenAlex

Key points Postsynaptic α 1 ‐ and α 2 ‐adrenoreceptors produce tonic vasoconstriction in resting and contracting skeletal muscle. Muscular contraction attenuates sympathetic vasoconstriction (functional sympatholysis). The blunting of sympathetic vasoconstriction during contraction has been mechanistically linked to a reduction in postsynaptic α 1 ‐ and α 2 ‐adrenoreceptor responsiveness and nitric oxide. We recently demonstrated that exercise training augmented sympathetic vasoconstrictor responsiveness and functional sympatholysis. Whether these vascular adaptations were mediated by changes to postsynaptic adrenoreceptors was not investigated. The present findings demonstrate that exercise training augmented α 2 ‐adrenoreceptor‐ mediated vasoconstriction in resting and contracting skeletal muscle. These data indicate that exercise training alters the relative contributions of α‐adrenoreceptors to sympathetic vasoconstriction in resting and contracting skeletal muscle and that the regulation of sympathetic vasoconstriction becomes more complex following exercise training. Abstract We hypothesized that exercise training (ET) would alter α 2 ‐adrenoreceptor‐mediated sympathetic vasoconstriction. Sprague‐Dawley rats ( n = 30) were randomized to sedentary (S), mild‐ (M) or heavy‐intensity (H) treadmill ET groups (5 days per week for 4 weeks). Following the ET component of the study, rats were anaesthetized, and instrumented for lumbar sympathetic chain stimulation, triceps surae muscle contraction and measurement of femoral vascular conductance (FVC). The percentage change of FVC in response to sympathetic stimulation was determined at rest and during contraction in control, α 2 blockade (yohimbine) and combined α 2 + nitric oxide (NO) synthase (NOS) blockade ( N ω ‐nitro‐ l ‐arginine methyl ester hydrochloride, l ‐NAME) conditions. ET augmented ( P &lt; 0.05) sympathetic vasoconstrictor responses at rest and during contraction. Yohimbine reduced ( P &lt; 0.05) the vasoconstrictor response in ET rats at rest (M: 2 Hz: 8 ± 2%, 5 Hz: 9 ± 4%; H: 2 Hz: 14 ± 5%, 5 Hz: 11 ± 6%) and during contraction (M: 2 Hz: 9 ± 2%, 5 Hz: 9 ± 5%; H: 2 Hz: 8 ± 3%, 5 Hz: 6 ± 6%) but did not change the response in S rats. The addition of l ‐NAME caused a larger increase ( P &lt; 0.05) in the vasoconstrictor response in ET than in S rats at rest (2 Hz: S: 8 ± 2%, M: 15 ± 3%, H: 23 ± 7%; 5 Hz: S: 8 ± 5%, M: 15 ± 3%, H: 17 ± 5%) and during contraction (2 Hz: S: 9 ± 3%, M: 18 ± 3%, H: 22 ± 6%; 5 Hz: S: 9 ± 5%, M: 22 ± 4%, H:26 ± 9%). Sympatholysis was greater ( P &lt; 0.05) in ET than in S rats. Blockade of α 2 ‐adrenoreceptors and NOS reduced ( P &lt; 0.05) sympatholysis in ET rats, but had no effect on sympatholysis in S rats. In conclusion, ET increased α 2 ‐mediated vasoconstriction at rest and during contraction.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.958
Threshold uncertainty score0.341

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0010.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.019
GPT teacher head0.247
Teacher spread0.228 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations21
Published2013
Admission routes1
Has abstractyes

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