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Record W2095362984 · doi:10.1158/1538-7445.am2013-3573

Abstract 3573: 17β-estradiol enhances hPygopus2 expression via ERα-SP1 promoter complexes.

2013· article· en· W2095362984 on OpenAlexaff
Youlian R. Tzenov, Phillip Andrews, Kenneth R. Kao

Bibliographic record

VenueCancer Research · 2013
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicEstrogen and related hormone effects
Canadian institutionsMemorial University of Newfoundland
Fundersnot available
KeywordsChromatin immunoprecipitationSp1 transcription factorMolecular biologyCell cyclePromoterBiologyEstrogen receptorTranscription factorEstrogen receptor alphaChemistryGene expressionCellGeneCancerBiochemistry

Abstract

fetched live from OpenAlex

Abstract Background: The chromatin remodeling protein Human Pygopus2 (hPYGO2) is widely overexpressed in, and required for the proliferation of several cancer cell lines derived from a variety of tumors. Because of the requirement of hPYGO2 for the G1/S cell cycle transition, its cell cycle dependent expression and its regulation by the Retinoblastoma protein in breast cancer (BC) cells, we hypothesized a fundamental link to the cell cycle. Thus, due to their predominant role in BC, we tested if 17β-estradiol (E2) would cause upregulation of hPYGO2 through Estrogen receptor alpha-SP1 transcription factor (ERα-SP1) promoter complexes.Methods: Eight ERα positive (ER+) and negative (ER-) BC cell lines were grown in low serum conditions and treated with ERα agonists and/or antagonists. mRNA levels were analyzed by Q-PCR and protein by immunoblot. ERα and SP1 promoter binding sites and DNA binding domains were functionally inactivated by site-directed mutagenesis. ERα and SP1 occupancy at the hPYGO2 promoter after treatment with various activators/inhibitors was analyzed by chromatin immunoprecipitation assays. Cell cycle progression was assessed by flow cytometry.Results: hPygopus2 mRNA and protein levels were higher in ER+ BC cells relative to ER- cells. ERα agonists induced, while antagonists inhibited, hPygopus2 mRNA and protein expression. ERα and SP1 occupancy at the hPYGO2 promoter required intact promoter binding sites and functional DNA binding domains. E2 increased, while Fulvestrant decreased, ERα binding to the hPYGO2 promoter while having no effect on SP1 binding. SP1 directed siRNA reduced SP1 occupancy at the hPYGO2 promoter and decreased hPygopus2 mRNA and protein expression in both ER+ and ER- cells. Treatment with betulinic acid, an SP1 inhibitor, resulted in cell cycle arrest in ER- BC cell lines.Conclusion: Our findings indicate that ERα and SP1 regulate hPygopus2 expression via promoter binding and that SP1 continues to play a role in hPYGO2 regulation in ER- cells, thereby revealing the potential efficacy of betulinic acid as a treatment for endocrine-disruptor resistant BC. Citation Format: Youlian R. Tzenov, Phillip G. Andrews, Kenneth R. Kao. 17β-estradiol enhances hPygopus2 expression via ERα-SP1 promoter complexes. [abstract]. In: Proceedings of the 104th Annual Meeting of the American Association for Cancer Research; 2013 Apr 6-10; Washington, DC. Philadelphia (PA): AACR; Cancer Res 2013;73(8 Suppl):Abstract nr 3573. doi:10.1158/1538-7445.AM2013-3573

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.004
Threshold uncertainty score0.013

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0040.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.031
GPT teacher head0.356
Teacher spread0.325 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2013
Admission routes1
Has abstractyes

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