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Mutational Analysis Reveals the Origin and Therapy-Driven Evolution of Recurrent Glioma

2013· article· en· 1,387 citations· W2115276344 on OpenAlex· 10.1126/science.1239947

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A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.

Canadian affiliationAn author listed a Canadian institution. This is the only route the usual frame has.

Machine scores (provisional)

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Opus teacher head0.011
GPT teacher head0.271
Teacher spread
0.260 · how far apart the two teachers sit on this one work
Validation status
score_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Abstract

Tumor recurrence is a leading cause of cancer mortality. Therapies for recurrent disease may fail, at least in part, because the genomic alterations driving the growth of recurrences are distinct from those in the initial tumor. To explore this hypothesis, we sequenced the exomes of 23 initial low-grade gliomas and recurrent tumors resected from the same patients. In 43% of cases, at least half of the mutations in the initial tumor were undetected at recurrence, including driver mutations in TP53, ATRX, SMARCA4, and BRAF; this suggests that recurrent tumors are often seeded by cells derived from the initial tumor at a very early stage of their evolution. Notably, tumors from 6 of 10 patients treated with the chemotherapeutic drug temozolomide (TMZ) followed an alternative evolutionary path to high-grade glioma. At recurrence, these tumors were hypermutated and harbored driver mutations in the RB (retinoblastoma) and Akt-mTOR (mammalian target of rapamycin) pathways that bore the signature of TMZ-induced mutagenesis.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

The record

Venue
Science
Topic
Cancer Genomics and Diagnostics
Field
Biochemistry, Genetics and Molecular Biology
Canadian institutions
Canada's Michael Smith Genome Sciences Centre
Funders
National Institute of Neurological Disorders and StrokeNational Institute of General Medical SciencesNational Cancer Institute
Keywords
TemozolomideGliomaExome sequencingMedicineOncologyMutationInternal medicineBiologyCancer researchGeneticsGene
Has abstract in OpenAlex
yes