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Record W2118594175 · doi:10.1186/1471-2466-13-64

Nitric oxide synthase polymorphisms, gene expression and lung function in chronic obstructive pulmonary disease

2013· article· en· W2118594175 on OpenAlexafffund
Farzian Aminuddin, Tillie‐Louise Hackett, Dorota Stefanowicz, Aabida Saferali, Peter D. Paré, Amund Gulsvik, Per Bakke, Michael H. Cho, Augusto A. Litonjua, David A. Lomas, Wayne H. Anderson, Terri H. Beaty, Edwin K. Silverman, Andrew J. Sandford

Bibliographic record

VenueBMC Pulmonary Medicine · 2013
Typearticle
Languageen
FieldMedicine
TopicChronic Obstructive Pulmonary Disease (COPD) Research
Canadian institutionsProvidence Health CareUniversity of British Columbia
FundersUniversity of Texas Health Science Center at San AntonioUniversity of California, San DiegoAgency for Healthcare Research and QualityNational Institutes of HealthMichael Smith Health Research BCMorehouse School of MedicineTemple UniversityCOPD FoundationGlaxoSmithKlineAstraZenecaCanadian Institutes of Health ResearchSunovionUniversity of MinnesotaBrigham and Women's HospitalCenters for Medicare and Medicaid ServicesUniversity of PittsburghNational Jewish HealthJohns Hopkins UniversityNational Heart, Lung, and Blood InstitutePfizerU.S. Department of Veterans Affairs
KeywordsMedicineNitric oxide synthasePulmonary diseaseLung functionGenePulmonary function testingGene expressionLungNitric oxideDiseaseNitric Oxide Synthase Type IIIBioinformaticsGeneticsImmunologyPathologyInternal medicineBiology

Abstract

fetched live from OpenAlex

BACKGROUND: Due to the pleiotropic effects of nitric oxide (NO) within the lungs, it is likely that NO is a significant factor in the pathogenesis of chronic obstructive pulmonary disease (COPD). The aim of this study was to test for association between single nucleotide polymorphisms (SNPs) in three NO synthase (NOS) genes and lung function, as well as to examine gene expression and protein levels in relation to the genetic variation. METHODS: One SNP in each NOS gene (neuronal NOS (NOS1), inducible NOS (NOS2), and endothelial NOS (NOS3)) was genotyped in the Lung Health Study (LHS) and correlated with lung function. One SNP (rs1800779) was also analyzed for association with COPD and lung function in four COPD case-control populations. Lung tissue expression of NOS3 mRNA and protein was tested in individuals of known genotype for rs1800779. Immunohistochemistry of lung tissue was used to localize NOS3 expression. RESULTS: For the NOS3 rs1800779 SNP, the baseline forced expiratory volume in one second in the LHS was significantly higher in the combined AG + GG genotypic groups compared with the AA genotypic group. Gene expression and protein levels in lung tissue were significantly lower in subjects with the AG + GG genotypes than in AA subjects. NOS3 protein was expressed in the airway epithelium and subjects with the AA genotype demonstrated higher NOS3 expression compared with AG and GG individuals. However, we were not able to replicate the associations with COPD or lung function in the other COPD study groups. CONCLUSIONS: Variants in the NOS genes were not associated with lung function or COPD status. However, the G allele of rs1800779 resulted in a decrease of NOS3 gene expression and protein levels and this has implications for the numerous disease states that have been associated with this polymorphism.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesMeta-epidemiology (narrow), Insufficient payload (model declined to judge)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.429
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0010.001
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0010.001
Science and technology studies0.0000.001
Scholarly communication0.0000.001
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0030.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.018
GPT teacher head0.273
Teacher spread0.255 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations19
Published2013
Admission routes2
Has abstractyes

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