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Record W2120318535 · doi:10.1002/art.24387

High‐density genotyping of STAT4 reveals multiple haplotypic associations with systemic lupus erythematosus in different racial groups

2009· article· en· W2120318535 on OpenAlexaff
Bahram Namjou, Andrea L. Sestak, Don Armstrong, Raphael Zidovetzki, Jennifer A. Kelly, Noam Jacob, Voicu Ciobanu, Kenneth M. Kaufman, Joshua O. Ojwang, Julie T. Ziegler, Francesco P. Quismorio, Andreas Reiff, Barry L. Myones, Joel M. Guthridge, Swapan K. Nath, Gail R. Bruner, Ruth Mehrian‐Shai, Earl D. Silverman, Marisa S. Klein‐Gitelman, Deborah McCurdy, Linda Wagner‐Weiner, James J. Nocton, Chaim Putterman, Sang‐Cheol Bae, Yun Jung Kim, Michelle Petri, John D. Reveille, Timothy J. Vyse, Gary S. Gilkeson, Diane L. Kamen, Marta E. Alarcón‐Riquelme, Patrick M. Gaffney, Kathy L. Moser, Joan T. Merrill, R. Hal Scofield, Judith A. James, Carl D. Langefeld, John B. Harley, Chaim O. Jacob

Bibliographic record

VenueArthritis & Rheumatism · 2009
Typearticle
Languageen
FieldMedicine
TopicSystemic Lupus Erythematosus Research
Canadian institutionsHospital for Sick Children
FundersNational Center for Research ResourcesNational Institute of Arthritis and Musculoskeletal and Skin DiseasesNational Institute of Allergy and Infectious DiseasesNational Institutes of HealthUniversity of AlabamaU.S. Department of Veterans AffairsUniversity of Southern CaliforniaNational Institute of Dental and Craniofacial ResearchLupus Research AllianceOklahoma Medical Research FoundationUniversity of Alabama at Birmingham
KeywordsHaplotypeSingle-nucleotide polymorphismSTAT4ImmunologyGenotypingGenetic associationAllele frequencyPathogenesisMedicineGeneticsAlleleOdds ratioGenotypeBiologyInternal medicineGene

Abstract

fetched live from OpenAlex

OBJECTIVE: Systemic lupus erythematosus (SLE) is the prototypical systemic autoimmune disorder, with complex etiology and a strong genetic component. Recently, gene products involved in the interferon pathway have been under intense investigation in terms of the pathogenesis of SLE. STAT-1 and STAT-4 are transcription factors that play key roles in the interferon and Th1 signaling pathways, making them attractive candidates for involvement in SLE susceptibility. METHODS: Fifty-six single-nucleotide polymorphisms (SNPs) across STAT1 and STAT4 on chromosome 2 were genotyped using the Illumina platform, as part of an extensive association study in a large collection of 9,923 lupus patients and control subjects from different racial groups. DNA samples were obtained from the peripheral blood of patients with SLE and control subjects. Principal components analyses and population-based case-control association analyses were performed, and the P values, false discovery rate q values, and odds ratios with 95% confidence intervals were calculated. RESULTS: We observed strong genetic associations with SLE and multiple SNPs located within STAT4 in different ethnic groups (Fisher's combined P = 7.02 x 10(-25)). In addition to strongly confirming the previously reported association in the third intronic region of this gene, we identified additional haplotypic association across STAT4 and, in particular, a common risk haplotype that is found in multiple racial groups. In contrast, only a relatively weak suggestive association was observed with STAT1, probably due to its proximity to STAT4. CONCLUSION: Our findings indicate that STAT4 is likely to be a crucial component in SLE pathogenesis in multiple racial groups. Knowledge of the functional effects of this association, when they are revealed, might improve our understanding of the disease and provide new therapeutic targets.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.001
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesMeta-epidemiology (narrow)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.046
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0010.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0000.001
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.015
GPT teacher head0.254
Teacher spread0.240 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations93
Published2009
Admission routes1
Has abstractyes

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