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Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS
Why is this work in the frame?
A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.
Canadian funderA Canadian agency funded it. The work may carry no Canadian affiliation at all.
No Canadian affiliation. An affiliation-only frame — the usual design — would never have seen this work. It is one of the works that make the case for inverting the frame.
Abstract
No abstract. This is not a gap in this database — OpenAlex has none either. 23.3% of the frame is in this state, and the screen finds HALF as much metaresearch here, so the absence is a measured bias rather than a missing field.
The record
- Venue
- Nature
- Topic
- Mitochondrial Function and Pathology
- Field
- Biochemistry, Genetics and Molecular Biology
- Canadian institutions
- —
- Funders
- Biotechnology and Biological Sciences Research CouncilNational Heart, Lung, and Blood InstituteMedical Research CouncilCanadian Institutes of Health ResearchBritish Heart Foundation
- Keywords
- Reactive oxygen speciesMitochondrial ROSReperfusion injuryMitochondrionIschemiaCitric acid cycleCell biologySuccinate dehydrogenaseBiologyIn vivoBiochemistryChemistryPharmacologyMetabolismMedicineInternal medicine
- Has abstract in OpenAlex
- no