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Record W2127489305 · doi:10.1161/atvb.34.suppl_1.2

Abstract 2: Elucidation of the Mechanism of Apolipoprotein(a)/Lipoprotein(a)-Induced Interleukin 8 Gene Expression: Insight into Potential Receptors and Signaling Pathways and the Role of Oxidized Phospholipids

2014· article· en· W2127489305 on OpenAlexaff
Corey A. Scipione, Sera Sayegh, Michael B. Boffa, Marlys L. Koschinsky

Bibliographic record

VenueArteriosclerosis Thrombosis and Vascular Biology · 2014
Typearticle
Languageen
FieldImmunology and Microbiology
TopicAtherosclerosis and Cardiovascular Diseases
Canadian institutionsUniversity of Windsor
Fundersnot available
KeywordsApolipoprotein BReceptorSignal transductionTLR2SecretionKringle domainBiologyLipoprotein(a)ChemistryMutationLipoproteinBiochemistryMolecular biologyCell biologyGeneRecombinant DNATLR4Cholesterol

Abstract

fetched live from OpenAlex

Elevated plasma Lipoprotein(a) (Lp(a)) levels are a risk factor for a variety of atherosclerotic disorders, although the exact role(s) of Lp(a) in atherogenesis remains poorly understood. It is clear, however, that the unique pathogenic effects attributed to Lp(a) are mediated by its apolipoprotein(a) moiety. Over the past decade, a growing body of evidence suggests that the harmful effects of apo(a) involve stimulation of inflammatory responses such as monocyte chemotaxis. It was previously reported that Lp(a), through apo(a), induces IL-8 gene expression by macrophages. The kringle V domain of apo(a) was implicated in this effect, along with oxidized phospholipids potentially present in this domain. In the current study, we found that a recombinant apo(a) species containing 17 kringle-IV domians (17K) elicited a dose-dependant increase in IL-8 mRNA abundance and protein secretion in THP-1 macrophages. This effect was significantly reduced by mutation of the strong lysine binding site (sLBS) in kringle IV type 10, but not by treatment with lysine analogue ε-ACA. Notably, this mutation eliminates the EO6-reactive oxidized phsopholipid in apo(a). Removal of kringle V did not reduce the ability of apo(a) to enhance IL-8 expression. Therefore, to examine the potential role of the modification of apo(a) by oxidized phospholipids, we used an siRNA knockdown approach to screen candidate receptors. We found that CD36 and TLR2, but not TLR4, are required for apo(a)-induced IL-8 expression. Specific MAPK pathway inhibitors were used to investigate the mechanism of IL-8 induction by apo(a): inhibitors of JNK and ERK1/2 abolished the effect of apo(a) whereas an inhibitor of p38 had no effect. Accordingly, apo(a) also stimulated an increase in JNK phosphorylation. To assess the role of downstream transcription factors, luciferase reporter gene experiments were conducted using the IL-8 promoter. Apo(a) induced expression of this reporter construct, as expected, and this induction was eliminated by mutation of known binding sites for NFκB or AP-1. Our results provide tantalizing mechanistic links between apo(a) lysine binding function, oxidative modification, and stimulation of intracellular signaling pathways through inflammation-linked receptors.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.004
Threshold uncertainty score0.014

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0040.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.011
GPT teacher head0.204
Teacher spread0.193 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2014
Admission routes1
Has abstractyes

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