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Record W2131617435 · doi:10.1002/humu.21446

Recurrence and variability of germline <i>EPCAM</i> deletions in Lynch syndrome

2011· article· en· W2131617435 on OpenAlexaff
Roland P. Kuiper, Lisenka E.L.M. Vissers, Ramprasath Venkatachalam, Daniëlle Bodmer, Eveline Hoenselaar, Monique Goossens, Aline Haufe, Eveline J. Kamping, Renée C. Niessen, Frans B.L. Hogervorst, B. Redeker, Carli M.J. Tops, Mariëlle van Gijn, Ans M.W. van den Ouweland, Nils Rahner, Verena Steinke, Philip Kahl, Elke Holinski‐Feder, Monika Morak, Matthias Kloor, Susanne Stemmler, Beate Betz, Pierre Hutter, David J. Bunyan, Sapna Syngal, Julie O. Culver, Tracy Graham, Tsun L. Chan, Irıs D. Nagtegaal, J. Han van Krieken, Hans K. Schackert, Ad Geurts van Kessel, Marjolijn J. L. Ligtenberg

Bibliographic record

VenueHuman Mutation · 2011
Typearticle
Languageen
FieldMedicine
TopicGenetic factors in colorectal cancer
Canadian institutionsHealth Sciences CentreSunnybrook Health Science Centre
FundersDeutsche KrebshilfeKWF KankerbestrijdingZonMw
KeywordsLynch syndromeMSH2BiologyNon-allelic homologous recombinationGeneticsBreakpointMLH1Epithelial cell adhesion moleculeGermlineMolecular biologyDNA mismatch repairGeneCancerDNA repairChromosomal translocationRecombinationGenetic recombination

Abstract

fetched live from OpenAlex

Recently, we identified 3' end deletions in the EPCAM gene as a novel cause of Lynch syndrome. These truncating EPCAM deletions cause allele-specific epigenetic silencing of the neighboring DNA mismatch repair gene MSH2 in tissues expressing EPCAM. Here we screened a cohort of unexplained Lynch-like families for the presence of EPCAM deletions. We identified 27 novel independent MSH2-deficient families from multiple geographical origins with varying deletions all encompassing the 3' end of EPCAM, but leaving the MSH2 gene intact. Within The Netherlands and Germany, EPCAM deletions appeared to represent at least 2.8% and 1.1% of the confirmed Lynch syndrome families, respectively. MSH2 promoter methylation was observed in epithelial tissues of all deletion carriers tested, thus confirming silencing of MSH2 as the causative defect. In a total of 45 families, 19 different deletions were found, all including the last two exons and the transcription termination signal of EPCAM. All deletions appeared to originate from Alu-repeat mediated recombination events. In 17 cases regions of microhomology around the breakpoints were found, suggesting nonallelic homologous recombination as the most likely mechanism. We conclude that 3' end EPCAM deletions are a recurrent cause of Lynch syndrome, which should be implemented in routine Lynch syndrome diagnostics.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.003
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.003

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.003
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.045
GPT teacher head0.295
Teacher spread0.250 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations152
Published2011
Admission routes1
Has abstractyes

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