MétaCan
Menu
Back to cohort
Record W2141865456 · doi:10.1093/brain/awq130

Common genetic variation and susceptibility to partial epilepsies: a genome-wide association study

2010· article· en· W2141865456 on OpenAlex
Dalia Kasperavičiūtė, Claudia B. Catarino, Erin L. Heinzen, Chantal Depondt, Gianpiero L. Cavalleri, Luís Otávio Sales Ferreira Caboclo, Sarah K. Tate, Jennifer Jamnadas-Khoda, Krishna Chinthapalli, Lisa M. Clayton, Kevin V. Shianna, Rodney A. Radtke, Mohamad A. Mikati, William B. Gallentine, Aatif M. Husain, Saud Alhusaini, David Leppert, Lefkos Middleton, Rachel A. Gibson, Michael R. Johnson, Paul M. Matthews, David A. Hosford, Kjell Heuser, Leslie Amos, Marcos Ortega, Dominik Zumsteg, Heinz Gregor Wieser, Bernhard J. Steinhoff, Günter Krämer, Jörg Hansen, Thomas Dorn, Anne-Mari Kantanen, Leif Gjerstad, Terhi Peuralinna, Dena Hernández, Kai Eriksson, Reetta Kälviäinen, Colin P. Doherty, Nicholas Wood, Massimo Pandolfo, John S. Duncan, Josemir W. Sander, Norman Delanty, David B. Goldstein, Sanjay M. Sisodiya

Why this work is in the frame

A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.

fundA Canadian funder is recorded on the work.
no affNo Canadian affiliation: this work is invisible to an affiliation-only frame.
No Canadian affiliation. An affiliation-only frame, the usual design, would never have seen this work. It is one of the works that make the case for inverting the frame.

Bibliographic record

VenueBrain · 2010
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicGenomics and Rare Diseases
Canadian institutionsnot available
FundersInstitute of GeneticsMedical Research CouncilUniversity College London Hospitals NHS Foundation TrustNational Institute for Health and Care ResearchHelsingin ja Uudenmaan SairaanhoitopiiriWellcome TrustHelsingin Yliopisto
KeywordsGenome-wide association studyHeritabilityGenetic associationBiologyGenetic variationMissing heritability problemGenetic architectureGeneticsGenetic predispositionOdds ratioSingle-nucleotide polymorphismGenotypePhenotypeMedicineGenePathology

Abstract

fetched live from OpenAlex

Partial epilepsies have a substantial heritability. However, the actual genetic causes are largely unknown. In contrast to many other common diseases for which genetic association-studies have successfully revealed common variants associated with disease risk, the role of common variation in partial epilepsies has not yet been explored in a well-powered study. We undertook a genome-wide association-study to identify common variants which influence risk for epilepsy shared amongst partial epilepsy syndromes, in 3445 patients and 6935 controls of European ancestry. We did not identify any genome-wide significant association. A few single nucleotide polymorphisms may warrant further investigation. We exclude common genetic variants with effect sizes above a modest 1.3 odds ratio for a single variant as contributors to genetic susceptibility shared across the partial epilepsies. We show that, at best, common genetic variation can only have a modest role in predisposition to the partial epilepsies when considered across syndromes in Europeans. The genetic architecture of the partial epilepsies is likely to be very complex, reflecting genotypic and phenotypic heterogeneity. Larger meta-analyses are required to identify variants of smaller effect sizes (odds ratio<1.3) or syndrome-specific variants. Further, our results suggest research efforts should also be directed towards identifying the multiple rare variants likely to account for at least part of the heritability of the partial epilepsies. Data emerging from genome-wide association-studies will be valuable during the next serious challenge of interpreting all the genetic variation emerging from whole-genome sequencing studies.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.236
Threshold uncertainty score0.380

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.006
GPT teacher head0.242
Teacher spread0.237 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it