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Record W2142907734 · doi:10.1016/j.ajhg.2015.02.009

A Large-Scale Genetic Analysis Reveals a Strong Contribution of the HLA Class II Region to Giant Cell Arteritis Susceptibility

2015· article· en· W2142907734 on OpenAlexaff
F. David Carmona, Sarah Mackie, Jose‐Ezequiel Martín, John Taylor, Augusto Vaglio, Stephen Eyre, Lara Bossini‐Castillo, Santos Castañeda, María C. Cid, José Hernández‐Rodríguez, Sergio Prieto‐González, Roser Solans, Marc Ramentol-Sintas, Marı́a-Francisca González-Escribano, Lourdes Ortiz‐Fernández, Inmaculada C. Morado, Javier Narváez, José A. Miranda‐Filloy, Lorenzo Beretta, Claudio Lunardi, Marco A. Cimmino, Davide Gianfreda, Daniele Santilli, Giuseppe A. Ramirez, Alessandra Soriano, Francesco Muratore, Giulia Pazzola, Olga Addimanda, Cisca Wijmenga, Torsten Witte, Jan Henrik Schirmer, Frank Moosig, Verena Schönau, André Franke, Øyvind Palm, Øyvind Molberg, Andreas P. Diamantopoulos, Simon Carette, David Cuthbertson, Lindsy Forbess, Gary S. Hoffman, Nader Khalidi, Curry L. Koening, Carol A. Langford, Carol A. McAlear, Larry W. Moreland, Paul A. Monach, Christian Pagnoux, Philip Seo, Robert Spiera, Antoine G. Sreih, Kenneth J. Warrington, Steven R. Ytterberg, Peter K. Gregersen, Colin Pease, Andrew Gough, Michael R. Green, Lesley Hordon, Stephen Jarrett, Richard A. Watts, Sarah Levy, Yusuf Patel, Sanjeet Kamath, Bhaskar DasGupta, Jane Worthington, Bobby P. C. Koeleman, Paul I. W. de Bakker, Jennifer H. Barrett, Carlo Salvarani, Peter A. Merkel, Miguel Á. González‐Gay, Ann W Morgan, Javier Martı́n, Agustín Martínez-Berriochoa, Ainhoa Unzurrunzaga, Ana Hidalgo-Conde, Ana Belén Madroñero-Vuelta, Antonio Fernández‐Nebro, M. Carmen Ordóñez-Cañizares, B. Escalante, Begoña Marí-Alfonso, B. Sopeña, Enrique Raya, Elena Grau, José A. Ruiz San Román, Eugenio de Miguel, FJ López-Longo, Lina Martínez, Carmen Gómez‐Vaquero, Benjamín Fernández‐Gutiérrez, Luis Rodríguez‐Rodríguez, J. Bernardino Díaz-López, Luis Caminal‐Montero, Aleida Martínez Zapico, Jordi Monfort, Laura Tío, Julio Sánchez-Martín, Juan José Alegre Sancho, Luís Sáez-Comet, Mercedes Pérez-Conesa, Marc Corbera‐Bellalta, María Jesús García-Villanueva, M.E. Fernández-Contreras, Olga Sánchez‐Pernaute, Ricardo Blanco, Norberto Ortego‐Centeno, Raquel Ríos-Fernández, José Luís Callejas-Rubio, P. Fanlo-Mateo, Víctor Martínez‐Taboada

Bibliographic record

VenueThe American Journal of Human Genetics · 2015
Typearticle
Languageen
FieldImmunology and Microbiology
TopicAtherosclerosis and Cardiovascular Diseases
Canadian institutionsMcMaster UniversitySt. Joseph’s Healthcare HamiltonMount Sinai Hospital
FundersNational Center for Advancing Translational SciencesNational Institute of Arthritis and Musculoskeletal and Skin DiseasesNational Center for Research ResourcesMedical Research CouncilDeutsche ForschungsgemeinschaftWellcome TrustAcademy of Medical SciencesInstituto de Salud Carlos IIINational Institute for Health and Care ResearchNational Institutes of HealthRare Diseases Clinical Research Network
KeywordsHuman leukocyte antigenGiant cell arteritisScale (ratio)Class (philosophy)GeneticsImmunologyBiologyGeographyMedicineVasculitisDiseaseComputer scienceCartographyPathologyAntigenArtificial intelligence

Abstract

fetched live from OpenAlex

We conducted a large-scale genetic analysis on giant cell arteritis (GCA), a polygenic immune-mediated vasculitis. A case-control cohort, comprising 1,651 case subjects with GCA and 15,306 unrelated control subjects from six different countries of European ancestry, was genotyped by the Immunochip array. We also imputed HLA data with a previously validated imputation method to perform a more comprehensive analysis of this genomic region. The strongest association signals were observed in the HLA region, with rs477515 representing the highest peak (p = 4.05 × 10(-40), OR = 1.73). A multivariate model including class II amino acids of HLA-DRβ1 and HLA-DQα1 and one class I amino acid of HLA-B explained most of the HLA association with GCA, consistent with previously reported associations of classical HLA alleles like HLA-DRB1(∗)04. An omnibus test on polymorphic amino acid positions highlighted DRβ1 13 (p = 4.08 × 10(-43)) and HLA-DQα1 47 (p = 4.02 × 10(-46)), 56, and 76 (both p = 1.84 × 10(-45)) as relevant positions for disease susceptibility. Outside the HLA region, the most significant loci included PTPN22 (rs2476601, p = 1.73 × 10(-6), OR = 1.38), LRRC32 (rs10160518, p = 4.39 × 10(-6), OR = 1.20), and REL (rs115674477, p = 1.10 × 10(-5), OR = 1.63). Our study provides evidence of a strong contribution of HLA class I and II molecules to susceptibility to GCA. In the non-HLA region, we confirmed a key role for the functional PTPN22 rs2476601 variant and proposed other putative risk loci for GCA involved in Th1, Th17, and Treg cell function.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.002
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.006

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.002
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.001
Bibliometrics0.0010.001
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.014
GPT teacher head0.245
Teacher spread0.231 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations178
Published2015
Admission routes1
Has abstractyes

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