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Record W2149444011 · doi:10.1158/1538-7445.am2013-265

Abstract 265: Mechanisms of Met-dependent tumorigenesis in models of triple negative breast cancer.

2013· article· en· W2149444011 on OpenAlexaff
Vanessa Y.C. Sung, Jennifer F. Knight, Morag Park

Bibliographic record

VenueCancer Research · 2013
Typearticle
Languageen
FieldMedicine
TopicCancer Cells and Metastasis
Canadian institutionsMcGill University
Fundersnot available
KeywordsBreast cancerCancer researchCancerBiologyCarcinogenesisHepatocyte growth factorTriple-negative breast cancerReceptor tyrosine kinaseReceptorSignal transductionCell biologyGenetics

Abstract

fetched live from OpenAlex

Abstract Human breast cancer is a heterogeneous disease encompassing multiple subtypes. The basal-like and claudin-low subtypes, collectively known as triple negative (TN) breast cancer, are predominantly negative for the expression of therapeutic targets (Her2 and estrogen receptors) and have poor prognosis. In human breast cancer, elevated levels of MET are correlated with TN subtypes and poor outcome, but the reasons for this are relatively unknown. The Met receptor tyrosine kinase (RTK) is a cell surface protein that is activated in response to the Hepatocyte Growth Factor (HGF). Upon stimulation, Met signaling pathways initiate a program of cell survival, migration, and invasive growth. To investigate the role of Met in tumourigenesis, we generated transgenic mice with mammary specific expression of an oncogenic variant of Met (MMTV-Metmut). These mice develop tumours with features of TN breast cancer. Tumour-initiating cells (TICs) have emerged as a key concept in prevailing models of tumour development. In breast and other solid tumours, where the bulk of the tumour mass consists of differentiated non-tumourigenic cells, TICs constitute a cell fraction of less differentiated tumourigenic cells, and have been shown to be highly resistant to radiation and chemotherapy. TICs are thus a key therapeutic target, yet little is known about TICs in TN breast cancer. Using tumoursphere assays, we have established the presence of TICs in tumours derived from MMTV-Metmut transgenic mice. MMTV-Metmut TICs have constitutively active Met. Interestingly, treatment with a small molecule inhibitor targeting Met can diminish both tumoursphere formation and proliferation. Thus, in TN breast cancers, elevated levels of Met signaling may promote TIC tumourigenicity. Here, mechanisms of Met-dependent TIC propagation and tumourigenicity will be addressed and supporting data presented. The molecular events that lead to human basal-like and claudin-low breast cancers are poorly understood. Validating a role for Met in TICs could identify key pathways involved in the pathogenesis of TN breast cancer, providing information for new therapeutic targets. Citation Format: Vanessa Y.C. Sung, Jennifer F. Knight, Morag Park. Mechanisms of Met-dependent tumorigenesis in models of triple negative breast cancer. [abstract]. In: Proceedings of the 104th Annual Meeting of the American Association for Cancer Research; 2013 Apr 6-10; Washington, DC. Philadelphia (PA): AACR; Cancer Res 2013;73(8 Suppl):Abstract nr 265. doi:10.1158/1538-7445.AM2013-265

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.007

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.097
GPT teacher head0.394
Teacher spread0.296 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2013
Admission routes1
Has abstractyes

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