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Cross-Disorder Genome-Wide Analyses Suggest a Complex Genetic Relationship Between Tourette’s Syndrome and OCD

2014· article· en· W2155276106 on OpenAlex

Why this work is in the frame

A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.

fundA Canadian funder is recorded on the work.
no affNo Canadian affiliation: this work is invisible to an affiliation-only frame.
No Canadian affiliation. An affiliation-only frame, the usual design, would never have seen this work. It is one of the works that make the case for inverting the frame.

Bibliographic record

VenueAmerican Journal of Psychiatry · 2014
Typearticle
Languageen
FieldPsychology
TopicObsessive-Compulsive Spectrum Disorders
Canadian institutionsnot available
FundersCilagDavid Geffen School of Medicine, University of California, Los AngelesNational Center for Advancing Translational SciencesNational Human Genome Research InstituteNational Institute on Drug AbuseNational Institute of Mental HealthSt. George's, University of LondonCanadian Institutes of Health ResearchEli Lilly CanadaDepartment of Psychiatry, University of TorontoStanley Center for Psychiatric Research, Broad InstituteUniversity of California, IrvineDirectorate for Biological SciencesCenters for Disease Control and PreventionMedical Research CouncilServierUniversidad Pontificia BolivarianaJulius-Maximilians-Universität WürzburgUniversität zu KölnOtsuka PharmaceuticalNational Institute of Neurological Disorders and StrokeUniversität ZürichH. Lundbeck A/SHospital for Sick ChildrenUniversidade de São PauloHebrew University of JerusalemBundesministerium für Bildung und ForschungUniversiteit van AmsterdamAstraZenecaEuropean CommissionVrije Universiteit AmsterdamUniversity of OxfordChild and Family Research InstituteFondation FondaMentalUniversidad de AntioquiaSanofiBroad InstituteUniversity of California, Los AngelesPfizerUniversità degli Studi di MilanoAllison Family FoundationJazz PharmaceuticalsDeutsche ForschungsgemeinschaftConselho Nacional de Desenvolvimento Científico e TecnológicoMcGill UniversityNational Institutes of HealthAmerican Academy of Child and Adolescent PsychiatryJane and Terry Semel Institute for Neuroscience and Human Behavior, University of California, Los AngelesUniversity of CincinnatiMinistero dell’Istruzione, dell’Università e della RicercaSchweizerischer Nationalfonds zur Förderung der Wissenschaftlichen ForschungYork UniversityEberhard Karls Universität TübingenUniversity of California, San DiegoJohns Hopkins UniversityCincinnati Children's Hospital Medical CenterF. Hoffmann-La RocheWayne State UniversityNational Cancer InstituteGrifolsUniversity College LondonUniversity of Southern CaliforniaBrigham and Women's HospitalMcLean HospitalUniversity of British ColumbiaEli Lilly and CompanyKoninklijke Nederlandse Akademie van WetenschappenUniversity of Illinois at Urbana-ChampaignMichael Smith Health Research BCRheinische Friedrich-Wilhelms-Universität BonnInternational OCD FoundationRijksuniversiteit GroningenDNA GenotekFP7 HealthUniversity of TorontoAmerican Psychiatric PublishingBristol-Myers SquibbTeva Pharmaceutical IndustriesNational Science FoundationUniversity of Cape TownMassachusetts General HospitalUniversity of California, San FranciscoGlaxoSmithKlineNational Institute on Alcohol Abuse and AlcoholismUniversity of New South WalesYale University
KeywordsGenome-wide association studyTourette syndromeSingle-nucleotide polymorphismTicsGenetic associationGenetic architectureGeneticsBiologyPhenotypePsychologyGeneNeurosciencePsychiatryGenotype

Abstract

fetched live from OpenAlex

OBJECTIVE: Obsessive-compulsive disorder (OCD) and Tourette's syndrome are highly heritable neurodevelopmental disorders that are thought to share genetic risk factors. However, the identification of definitive susceptibility genes for these etiologically complex disorders remains elusive. The authors report a combined genome-wide association study (GWAS) of Tourette's syndrome and OCD. METHOD: The authors conducted a GWAS in 2,723 cases (1,310 with OCD, 834 with Tourette's syndrome, 579 with OCD plus Tourette's syndrome/chronic tics), 5,667 ancestry-matched controls, and 290 OCD parent-child trios. GWAS summary statistics were examined for enrichment of functional variants associated with gene expression levels in brain regions. Polygenic score analyses were conducted to investigate the genetic architecture within and across the two disorders. RESULTS: Although no individual single-nucleotide polymorphisms (SNPs) achieved genome-wide significance, the GWAS signals were enriched for SNPs strongly associated with variations in brain gene expression levels (expression quantitative loci, or eQTLs), suggesting the presence of true functional variants that contribute to risk of these disorders. Polygenic score analyses identified a significant polygenic component for OCD (p=2×10(-4)), predicting 3.2% of the phenotypic variance in an independent data set. In contrast, Tourette's syndrome had a smaller, nonsignificant polygenic component, predicting only 0.6% of the phenotypic variance (p=0.06). No significant polygenic signal was detected across the two disorders, although the sample is likely underpowered to detect a modest shared signal. Furthermore, the OCD polygenic signal was significantly attenuated when cases with both OCD and co-occurring Tourette's syndrome/chronic tics were included in the analysis (p=0.01). CONCLUSIONS: Previous work has shown that Tourette's syndrome and OCD have some degree of shared genetic variation. However, the data from this study suggest that there are also distinct components to the genetic architectures of these two disorders. Furthermore, OCD with co-occurring Tourette's syndrome/chronic tics may have different underlying genetic susceptibility compared with OCD alone.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesMeta-epidemiology (narrow)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.006
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0010.001
Science and technology studies0.0000.001
Scholarly communication0.0000.000
Open science0.0010.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.025
GPT teacher head0.345
Teacher spread0.320 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it