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Identification of Oxidative Stress and Toll-like Receptor 4 Signaling as a Key Pathway of Acute Lung Injury
Why is this work in the frame?
A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.
Canadian affiliationAn author listed a Canadian institution. This is the only route the usual frame has.
Canadian funderA Canadian agency funded it. The work may carry no Canadian affiliation at all.
Abstract
No abstract. This is not a gap in this database — OpenAlex has none either. 23.3% of the frame is in this state, and the screen finds HALF as much metaresearch here, so the absence is a measured bias rather than a missing field.
The record
- Venue
- Cell
- Topic
- Immune Response and Inflammation
- Field
- Immunology and Microbiology
- Canadian institutions
- St. Michael's HospitalUniversity of TorontoLawson Health Research InstituteWestern University
- Funders
- Defense Threat Reduction AgencySchweizerischer Nationalfonds zur Förderung der Wissenschaftlichen ForschungCanadian Institutes of Health ResearchEuropean CommissionUniversity of Pittsburgh
- Keywords
- TRIFTLR4Oxidative stressBiologyImmunologyInnate immune systemInfluenza A virus subtype H5N1LungToll-like receptorReceptorSignal transductionInflammationImmune systemMedicineCell biologyInternal medicineVirusBiochemistry
- Has abstract in OpenAlex
- no