MétaCan
Menu
Back to cohort
Record W2178721006 · doi:10.3389/fneur.2015.00232

Pathogenic microRNAs Common to Brain and Retinal Degeneration; Recent Observations in Alzheimer’s Disease and Age-Related Macular Degeneration

2015· review· en· W2178721006 on OpenAlexaff
James M. Hill, Aileen I. Pogue, Walter J. Lukiw

Bibliographic record

VenueFrontiers in Neurology · 2015
Typereview
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicMicroRNA in disease regulation
Canadian institutionsNorgen Biotek Corporation (Canada)
FundersNational Eye InstituteLouisiana Biomedical Research NetworkResearch to Prevent BlindnessNational Institute on AgingNational Institutes of HealthAlzheimer's Association
KeywordsNeurodegenerationMacular degenerationDegeneration (medical)Retinal degenerationNeuroscienceDiseaseRetinalNeuronal degenerationMedicineAlzheimer's diseasePathologyOphthalmologyBiology

Abstract

fetched live from OpenAlex

MicroRNAs (miRNAs), ~22 nt single-stranded non-coding RNAs (ncRNAs) abundant in the human brain and retina, have emerged as significant post-transcriptional regulators of messenger RNA (mRNA) abundance and complexity in the human central nervous system (CNS) in aging, health, and disease.Of the 2050 different miRNAs in the human body so far identified, only about 25-30 are abundant in either the brain or the retina, underscoring the high selection pressure carried by RNA sequences located within these select ncRNAs (1-7).It is noteworthy to point out that: (i) that brain neocortex and retina share a common neuroectodermal origin; (ii) that brain and retina share a subfamily of specific miRNA species; and (iii) that the multilayered assemblies of both neural and retinal cells are targeted by pathogenic processes that drive progressive pro-inflammatory neurodegeneration (5-9).Indeed, pathologically upregulated miRNAs common to both the prototypic age-related inflammatory degeneration of the brain in Alzheimer's disease (AD) and of the retina in age-related macular degeneration (AMD) appear to be associated with deficits in the expression of messenger RNA (mRNA) and gene families involved in the innate-immune response, inflammation, neurotrophism, synaptogenesis, and amyloidogenesis (Figure 1).In this "Opinion" paper for the Frontiers in Neurology Special Research Topic, we will highlight some of the most recent work in this research area, with emphasis on a family of five up-regulated pro-inflammatory miRNAs -miRNA-9, miRNA-34a, miRNA-125b, miRNA-146a, and miRNA-155 -that are emerging as key mechanistic contributors to the AD and AMD process.Homeostatic levels of specific miRNAs are natural indicators of neurological health of both the brain and retina (2-10, 31).Recently, multiple independent neurological research laboratories have provided evidence for the up-regulation of a small group of five inducible miRNAs in age-related diseases involving a progressive inflammatory degeneration.That these five miRNAs -miRNA-9, miRNA-34a, miRNA-125b, miRNA-146a, and miRNA-155 -are up-regulated in both AD and AMD underscores the concept that the brain and retina share common pathological signaling of a preexisting subfamily of miRNAs that individually contribute to various aspects of neurodegenerative

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Not applicable · Consensus signal: none
GenreCandidate signal: Review · Consensus signal: Review
Teacher disagreement score0.001
Threshold uncertainty score0.003

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0010.001
Science and technology studies0.0000.001
Scholarly communication0.0010.001
Open science0.0000.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.039
GPT teacher head0.294
Teacher spread0.255 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designNot applicable
Domainnot available
GenreReview

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations30
Published2015
Admission routes1
Has abstractyes

Explore more

Same venueFrontiers in NeurologySame topicMicroRNA in disease regulationFrench-language works237,207