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Record W2216910951 · doi:10.1152/ajprenal.00169.2015

G protein α<sub>12</sub>(Gα<sub>12</sub>) is a negative regulator of kidney injury molecule-1-mediated efferocytosis

2015· article· en· W2216910951 on OpenAlexafffund
Ola Z. Ismail, Xizhong Zhang, Joseph V. Bonventre, Lakshman Gunaratnam

Bibliographic record

VenueAmerican Journal of Physiology-Renal Physiology · 2015
Typearticle
Languageen
FieldImmunology and Microbiology
TopicPhagocytosis and Immune Regulation
Canadian institutionsLawson Health Research InstituteWestern University
FundersNational Institute of Diabetes and Digestive and Kidney DiseasesGovernment of CanadaLawson Health Research InstituteCanadian Institutes of Health ResearchBiogen
KeywordsEfferocytosisRHOACell biologyApoptosisGene silencingBiologyChemistrySignal transductionCancer researchBiochemistryMacrophageIn vitro

Abstract

fetched live from OpenAlex

Kidney injury molecule-1 (KIM-1) is a receptor for the “eat me” signal, phosphatidylserine, on apoptotic cells. The specific upregulation of KIM-1 by injured tubular epithelial cells (TECs) enables them to clear apoptotic cells (also known as efferocytosis), thereby protecting from acute kidney injury. Recently, we uncovered that KIM-1 binds directly to the α-subunit of heterotrimeric G 12 protein (Gα 12 ) and inhibits its activation by reactive oxygen species during renal ischemia-reperfusion injury (Ismail OZ, Zhang X, Wei J, Haig A, Denker BM, Suri RS, Sener A, Gunaratnam L. Am J Pathol 185: 1207–1215, 2015). Here, we investigated the role that Gα 12 plays in KIM-1-mediated efferocytosis by TECs. We showed that KIM-1 remains bound to Gα 12 and suppresses its activity during phagocytosis. When we silenced Gα 12 expression using small interefering RNA, KIM-1-mediated engulfment of apoptotic cells was increased significantly; in contrast overexpression of constitutively active Gα 12 ( QLGα 12 ) resulted in inhibition of efferocytosis. Inhibition of RhoA, a key effector of Gα 12 , using a chemical inhibitor or expression of dominant-negative RhoA, had the same effect as inhibition of Gα 12 on efferocytosis. Consistent with this, silencing Gα 12 suppressed active RhoA in KIM-1-expressing cells. Finally, using primary TECs from Kim-1 +/+ and Kim-1 −/− mice, we confirmed that engulfment of apoptotic cells requires KIM-1 expression and that silencing Gα 12 enhanced efferocytosis by primary TECs. Our data reveal a previously unknown role for Gα 12 in regulating efferocytosis and that renal TECs require KIM-1 to mediate this process. These results may have therapeutic implications given the known harmful role of Gα 12 in acute kidney injury.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.001
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesMeta-epidemiology (narrow), Science and technology studies
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.023
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0010.001
Meta-epidemiology (narrow)0.0010.001
Meta-epidemiology (broad)0.0030.001
Bibliometrics0.0010.001
Science and technology studies0.0000.003
Scholarly communication0.0000.000
Open science0.0010.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.010
GPT teacher head0.230
Teacher spread0.220 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations25
Published2015
Admission routes2
Has abstractyes

Explore more

Same venueAmerican Journal of Physiology-Renal PhysiologySame topicPhagocytosis and Immune RegulationFrench-language works237,207