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Record W2245092729 · doi:10.82308/25940

Defining the role of different KRAS effectors in the initiation and progression of lung cancer

2013· article· en· W2245092729 on OpenAlexaboutno aff
Guillaume Vandal

Bibliographic record

VenueeScholarship@McGill (McGill) · 2013
Typearticle
Languageen
FieldImmunology and Microbiology
Topicinterferon and immune responses
Canadian institutionsnot available
FundersNational Institutes of Health
KeywordsKRASMAPK/ERK pathwayPI3K/AKT/mTOR pathwayCancer researchMEK inhibitorBiologyEffectorCancerLung cancerMutantCarcinogenesisSignal transductionMedicineCell biologyGeneticsOncologyGeneColorectal cancer

Abstract

fetched live from OpenAlex

Lung cancer is currently the most deadly malignancy in Canada, accounting for 27% of all cancer-related deaths. Over 70% of patients with non-small cell lung cancer (NSCLC) are diagnosed at a late stage, with a 5-year survival below 10%. In NSCLC, the two oncogenes that are most frequently mutated are the EGFR and KRAS genes. While targeted therapies have been developed for patients with EGFR mutations, oncogenic KRAS mutations are so far not druggable. KRAS is a small GTPase that acts as an on/off switch to activate multiple signalling pathways, including the PI3K/Akt pathway, the Raf-Mek-Erk pathway and the RalGDS/Ral pathway. In the BrafCA mouse model of lung tumourigenesis, it was shown that the Cre-mediated expression of BrafV600E, activating the Raf-Mek-Erk pathway, causes the formation of adenomas that undergo widespread senescence at the benign stage. However, oncogenic KRAS mutations in mice cause adenocarcinomas, which suggests that other pathways activated by KRAS cooperate with sustained RAF-MEK-ERK signalling to bypass the oncogene-induced senescence proliferation arrest. To elucidate which pathways may cooperate with the Raf-Mek-Erk pathway to lead to lung adenocarcinomas, I created four effector domain mutants of KRASV12 (S35, G37, E38 and C40). The S35 and E38 mutants bind to Raf proteins but not PI3K or RalGDS; the G37 mutant binds to RalGDS and not Raf or PI3K and the C40 mutant is specific to PI3K. I designed lentiviral vectors that code for the KRAS mutants (V12, V12/S35, V12/G37, V12/E38 or V12/C40), or eGFP as a negative control, bicistronically with the Cre recombinase. These lentiviruses were used to infect BrafCA/+ and wild-type mice. The biggest tumours seen in BrafCA/+ mice received the KRASV12 virus, followed closely by KRASV12/C40, suggesting that the PI3K and Raf-Mek-Erk pathways cooperate to increase tumour growth. There was a significant decrease in tumour penetrance in all conditions where any KRAS mutant was present compared to the eGFP control, suggesting that KRAS may directly activate effectors with tumour suppressive functions. Moreover, tumours in wild-type mice were only seen with KRASV12 expression.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.004

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0010.001
Open science0.0000.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.006
GPT teacher head0.244
Teacher spread0.238 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2013
Admission routes1
Has abstractyes

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