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Mevalonate Depletion Impairs Survival of Primary Human Mesenchymal Stem Cells through Down Regulation of NF-κB

2012· article· en· W2315701606 on OpenAlexaff
Y. Li, Kiranjit K Sran, Melanie Ngo, Rakesh C. Arora, Lorrie A. Kirshenbaum, Darren H. Freed

Bibliographic record

VenueTransplantation · 2012
Typearticle
Languageen
FieldMedicine
TopicTransplantation: Methods and Outcomes
Canadian institutionsUniversity of Manitoba
Fundersnot available
KeywordsAtorvastatinViability assaySimvastatinPravastatinMesenchymal stem cellNeointimaHMG-CoA reductaseProgenitor cellPharmacologyWestern blotLovastatinMevalonate pathwayBone marrowStem cellMedicineCancer researchChemistryBiologyImmunologyCell biologyCellReductaseCholesterolInternal medicineBiochemistry

Abstract

fetched live from OpenAlex

Background: Circulating progenitor cells of bone marrow origin have been implicated to neointima formation and constructive remodeling in vascular injury and transplant cardiac allograft vasculopathy (CAV). Hydroxymethylglutaryl-Coenzyme A (HMG-CoA) reductase inhibitors (“statins”) have been shown to slow the progression of CAV and improve patient survival, which was presumed to be as a result of altered lipid metabolism. Statins also exhibit other positive pleiotropic properties such as modulation of inflammatory response via a mechanism independent of cholesterol reduction. We examined the effect of HMG-CoA reductase inhibition with three statins (atorvastatin, simvastatin and pravastatin) on the viability of primary human bone marrow derived mesenchymal stem cells (MSC) in culture. Methods: The protocol received IRB approval. MSC were isolated from bone marrow collected from the sternum of patients undergoing open heart surgery and were cultured in standard conditions. Cells were treated with atorvastatin, simvastatin or pravastatin 0.1, 1.0 or 10 μM ± mevalonate for 48 or 96 hours. Optical MTT assay was performed to assess cell viability. Viability was also assessed based on physical and biochemical properties of the cell using live-dead assay. NF-κB p65 expression was assessed by western blot. Rescue of NF- κB pathway function was achieved through overexpression of Ikk-β with an adenoviral vector. Results: Treatment of MSC with 0.1, 1 or 10 μM atorvastatin or simvastatin resulted in progressively reduced cell viability up to 50% in a time and dose dependent manner which was associated with a corresponding progressive decline in NF-κB p65 expression. Viability could be rescued by coincubation with mevalonate or pretreatment with Ikk-β adenoviral vector. Treatment with pravastatin did not show any effect on cell viability or NF-κB p65 expression. Conclusions: Mevalonate depletion through HMG-CoA reductase inhibition impairs the viability of primary human MSC in culture through NF-κB p65 depletion. This additional pleiotropic effect of statins may provide an insight into the benefits of this therapy independent of its effect on lipid metabolism. The lack of effect on MSC viability by pravastatin could be due to the hydrophilic nature of the molecule.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.005

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.048
GPT teacher head0.320
Teacher spread0.272 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2012
Admission routes1
Has abstractyes

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