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Record W2316259484 · doi:10.1158/1538-7445.am10-lb-271

Abstract LB-271: The biological role of the transcription factor Kaiso in breast tumorigenesis

2010· article· en· W2316259484 on OpenAlexaff
Sonali Weerawardane, Juliet M. Daniel

Bibliographic record

VenueCancer Research · 2010
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicWnt/β-catenin signaling in development and cancer
Canadian institutionsMcMaster University
Fundersnot available
KeywordsWnt signaling pathwayCarcinogenesisCancer researchBiologyCell biologyEpithelial–mesenchymal transitionTranscription factorSignal transductionCancerMetastasis

Abstract

fetched live from OpenAlex

Abstract Introduction: The canonical Wnt signalling pathway regulates various cellular processes (e.g. proliferation, differentiation, apoptosis) and leads to tumorigenesis when aberrantly activated. -catenin, the key downstream effector in Wnt signalling, translocates to the nucleus upon Wnt pathway activation, and activates target genes involved in tumorigenesis. The transcription factor Kaiso is a novel BTB/POZ (Broad complex, Tramtrak, Bric à brac/Pox virus and zinc finger) zinc finger (POZ-ZF) protein that specifically interacts with the cell adhesion cofactor p120ctn. Recent studies from our lab and others have revealed that Kaiso negatively regulates the canonical Wnt pathway by repressing activation of Wnt/ -catenin target genes. Some studies implicate Kaiso as a tumor suppressor (decreased expression in breast, ovarian and colon tumors), and others implicate Kaiso in tumor initiation (Kaiso-deficient mice are resistant to intestinal cancer). The goal of this study is to elucidate the biological role of Kaiso in breast tumorigenesis using various cellular assays. Methods: To assess Kaiso's role in breast cancer, Kaiso was misexpressed in MCF7 (human breast adenocarcinoma) cell lines. Stable Kaiso overexpressing and depleted cell lines were generated and assessed for cell proliferation and cell migration using focus formation, wound healing and matrigel invasion assays. Western blot analysis was performed to assess the effects of Kaiso misexpression on p120 and E-cadherin as well as epithelial-mesenchymal transition markers such as N-cadherin, Snail, Twist and Vimentin. The subcellular localization of Kaiso and its putative target genes in vivo was examined using immunohistochemistry (IHC) analysis of matched pairs of human normal and tumor breast tissue. Results: Focus formation assays revealed increased transformation by cells over-expressing Kaiso, suggesting an oncogenic role for Kaiso in cell culture. Preliminary wound healing and matrigel assays show increased cell motility and migration respectively in Kaiso over-expressing cells compared to parental control cells, thereby suggesting a role for Kaiso in the epithelial-mesenchymal transition. IHC analysis revealed predominant nuclear Kaiso expression in lobular and ductal breast carcinomas in breast tissues. These data support Kaiso's potential role as an oncogene in human breast cancer. Citation Format: {Authors}. {Abstract title} [abstract]. In: Proceedings of the 101st Annual Meeting of the American Association for Cancer Research; 2010 Apr 17-21; Washington, DC. Philadelphia (PA): AACR; Cancer Res 2010;70(8 Suppl):Abstract nr LB-271.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.004
Threshold uncertainty score0.012

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0040.002

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.044
GPT teacher head0.338
Teacher spread0.294 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2010
Admission routes1
Has abstractyes

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