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Record W2320483694 · doi:10.1055/s-2006-943689

NOVEL SHORT-CHAIN ACYL-COA DEHYDROGENASE GENE MUTATION (319 C>T) PRESENTS WITH HYPOTONIA, DEVELOPMENTAL DELAY, MULTICORE MYOPATHY AND CLINICAL HETEROGENEITY AND IS CANDIDATE FOUNDER MUTATION IN ASHKENAZI JEWS

2006· article· en· W2320483694 on OpenAlexaff
Ingrid Tein, O Elpeleg, Bruria Ben‐Zeev, T. Lerman‐Sagie, N. Gregersen

Bibliographic record

VenueNeuropediatrics · 2006
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicMetabolism and Genetic Disorders
Canadian institutionsHospital for Sick Children
Fundersnot available
KeywordsHypotoniaMedicineGeneticsMutationFounder effectExome sequencingMyopathyCompound heterozygosityPediatricsGeneBiologyInternal medicineAlleleHaplotype

Abstract

fetched live from OpenAlex

Objectives: To characterize the demographic, clinical and biochemical phenotypic spectrum of children with short-chain acyl-CoA dehydrogenase (SCAD) deficiency due to the 319C >T mutation. Methods: Affected children were identified by elevated butyrylcarnitine on serum acylcarnitines (FAB-tandem MS), ethylmalonic aciduria on urine organic acids (GCMS) and/or deficient SCAD activity in cultured skin fibroblasts or biopsied muscle (ETF reduction assay). Genomic DNA was analyzed by PCR of 5'-flanking promoter-region & each of 10 SCAD gene exons in patients & in exon 3 of Ashkenazi Jewish controls.8. Results: We report 7 children (4 male, 3 female), 3 homozygous for the 319 C>T mutation and 4 who are compound heterozygous for the 319C>T and 625G>A mutations. All 7 are of Ashkenazi Jewish origin in which group, on screening, we found a high 319 C>T heterozygote frequency of 1:15 suggesting that this may be a founder mutation with a predicted homozygous birth rate of 1:780 or due to selective advantage. The clinical phenotype is variable with onset from birth to early childhood. Common features include hypotonia (6/7), developmental delay (5/6), myopathy (4/7) characterized by multiminicore changes in two & lipid storage in one, facial weakness (3/7), lethargy (3/7) & congenital abnormalities (3/7). One female with multicore myopathy had progressive external ophthalmoplegia, ptosis and cardiomyopathy with pneumonia and respiratory failure. Two compound heterozygous brothers presented with psychosis, optic atrophy, pyramidal signs, & multifocal white matter abnormalities on MRI brain suggesting additional genetic factors; one had cerebellar ataxia. Biochemical analysis revealed ethylmalonic aciduria (6/6), methylsuccinic aciduria (3/4), elevated butyrylcarnitine (3/5), decreased butyrate oxidation in lymphoblasts (2/4) and decreased SCAD activity in fibroblasts or muscle (3/3). Expression studies of 319 C>T mutation in isolated mouse liver mitochondria confirmed this to be disease causing. 625G>A is a common variant conferring disease susceptibility. Conclusion: We conclude that the wide clinical and biochemical phenotypic variability of this 319C>T mutation suggests that this is a complex multifactorial/polygenic condition that should be screened for in individuals with multicore myopathy, particularly among the Ashkenazi population.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Case report · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.007

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.001
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0010.000
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.014
GPT teacher head0.263
Teacher spread0.248 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designCase report
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2006
Admission routes1
Has abstractyes

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