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Record W2345513794 · doi:10.1161/atvb.35.suppl_1.414

Abstract 414: Key Role of STAT4 Deficiency in the Hematopoietic Compartment on Insulin Resistance and Adipose Tissue Inflammation

2015· article· en· W2345513794 on OpenAlexaff
Anca D. Dobrian, Kaiwen Ma, Lindsey Glenn, Margaret Hatcher, Bronson A. Haynes, Eric J. Lehrer, Jerry L Nalder

Bibliographic record

VenueArteriosclerosis Thrombosis and Vascular Biology · 2015
Typearticle
Languageen
FieldMedicine
TopicAdipokines, Inflammation, and Metabolic Diseases
Canadian institutionsHatch (Canada)
Fundersnot available
KeywordsSTAT4Adipose tissueEndocrinologyInternal medicineInflammationInsulin resistanceAdipocyteHaematopoiesisBiologyBone marrowCD8MedicineInsulinImmunologyImmune systemStem cellApoptosisstatBiochemistryCell biology

Abstract

fetched live from OpenAlex

Adipose tissue inflammation is a hallmark of obesity and contributes to insulin resistance (IR). Recent data from our lab showed that STAT4 global deficiency reduces inflammation and improves insulin sensitivity in obese mice. STAT4 is expressed in adipocytes and cells of hematopoietic lineage. The objective of this study was to determine the contribution of selective STAT4 deficiency in adipocyte vs hematopoietic cells to IR and adipose tissue inflammation. For this purpose we sub-lethally irradiated Stat-4 -/- C57Bl6 mice and reconstituted them with bone marrow cells (BMC) from Stat-4 +/+ C57Bl6 congenic donors to restore STAT4 expression in BM- derived cells (STAT4-/- recipients). These mice were compared to irradiated C57Bl6 wild-type mice reconstituted with Stat-4 -/- C57Bl6 BMCs to produce STAT4 deletion selectively in BM-derived cells (STAT4+/+ recipients). Mice received a HFD (60%kcal fat) for 12 weeks (n=7 mice/group). Body weights, plasma cholesterol, triglycerides and free fatty acids were not different between the STAT4+/+ and STAT4-/- recipients. However the STAT4-/- recipients had significantly increased fasted plasma glucose. Congruent with this data the IP-ITT and GTT showed significantly higher area under the curve for the STAT4-/- recipients indicating increased glucose intolerance and insulin resistance compared to STAT4+/+ recipients. Adipose tissue immune cell composition was determined by flow cytometry. The total number of CD45+ lymphocytes was significantly higher in the STAT4-/- recipients compared to STAT4+/+ recipients. Also, the numbers of CD3+CD4+ and CD3+CD8+ cells were significantly higher in the STAT4-/- recipient group. Adipocyte size was significantly higher and the number of crown-like structures was significantly increased in the STAT4 -/- recipients indicating adipocyte hypertrophy and increased macrophage infiltration. Following in vivo insulin stimulation, activation of IR and IRS-1 was increased in liver and muscle in the STAT4+/+ recipient group suggesting improved insulin signaling in the mice that received STAT4-/- BMCs. These studies provide clear evidence for the importance of STAT4 in hematopoietic cells in regulating IR and adipose tissue inflammation in obesity.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.007
Threshold uncertainty score0.023

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0070.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.034
GPT teacher head0.289
Teacher spread0.254 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2015
Admission routes1
Has abstractyes

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