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7A.02

2015· article· fr· W2398139883 on OpenAlexaff
Marek Jankowski, Éric Plante, Ahmed Ménaouar, J.‐L. Chiasson, Jolanta Gutkowska

Bibliographic record

VenueJournal of Hypertension · 2015
Typearticle
Languagefr
FieldPsychology
TopicNeuroendocrine regulation and behavior
Canadian institutionsCentre Hospitalier de l’Université de Montréal
Fundersnot available
KeywordsMedicineInternal medicineEndocrinologyResistinDiabetic cardiomyopathyCardiac function curveAdiponectinHeart failureDiabetes mellitusHyperinsulinemiaCardioprotectionInsulin resistanceCardiomyopathyMyocardial infarction

Abstract

fetched live from OpenAlex

Objective: Obesity and diabetes enhance the risk of developing cardiovascular diseases and heart failure. The heart/cardiac oxytocin (OT) system was discovered by our group and shown to regulate cardiovascular cell survival pathways and cardioprotection. OT is also involved in the regulation of cardiac energy metabolism and cardiac OT receptor is downregulated in diabetes. Our hypothesis is that OT prevents development of diabetic cardiomyopathy. We evaluated whether chronic treatment with OT could prevent the metabolic and cardiac abnormalities associated with diabetes and obesity using the db/db mice. Design and method: Four-week-old C57BL/KsJ-db/db obese diabetic mice (db/db) and their lean control littermates (db/+) were treated with OT (125 ng/kg/h) or saline during 12 weeks (n = 10/group). Serial blood and tomography analysis were performed. Cardiac function was determined by echocardiography, and biochemical and histological heart and fat analysis were also performed. Results: Compared to db/+ mice, the saline-treated db/db mice developed obesity, hyperglycemia and hyperinsulinemia. These mice also exhibited a deficient cardiac OT/natriuretic system and developed systolic and diastolic dysfunction resulting from cardiomyocytes hypertrophy, fibrosis and apoptosis. These abnormalities were associated with increased ROS production, inflammation and suppressed AMP-kinase signaling pathway. The db/db mice displayed reduced serum levels of adiponectin and adipsin and elevated resistin. OT treatment increased circulating OT levels, significantly reduced serum resistin, body fat accumulation (19%: p < 0.001), fasting blood glucose levels by (23%; p < 0.001), and improved glucose tolerance and insulin sensitivity. OT also normalized cardiac OT receptors, ANP and BNP expressions and prevented systolic and diastolic dysfunction as well as cardiomyocytes hypertrophy, fibrosis and apoptosis. Furthermore, OT reduced cardiac oxidative stress and inflammation, and normalized the AMP-activated protein kinase signaling pathway. The complete normalization of cardiac structure and function by OT treatment in db/db mice contrasted with only partial improvement of hyperglycemia and hyperinsulinemia. Conclusions: The results indicate that chronic treatment with OT partially improves glucose and fat metabolism, reverses abnormal cardiac structural remodeling, preventing cardiac dysfunction in db/db mice. These observations clearly suggest a potential role for OT in replacement therapy for the prevention of cardiovascular complications of diabetes and obesity.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.002
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesInsufficient payload (model declined to judge)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Not applicable · Consensus signal: none
GenreCandidate signal: Other · Consensus signal: Other
Teacher disagreement score0.102
Threshold uncertainty score0.000

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.002
Meta-epidemiology (narrow)0.0020.001
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0020.001
Science and technology studies0.0010.001
Scholarly communication0.0060.002
Open science0.0020.003
Research integrity0.0030.003
Insufficient payload (model declined to judge)0.8980.901

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.210
GPT teacher head0.371
Teacher spread0.161 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

Study designNot applicable
Domainnot available
GenreOther

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations1
Published2015
Admission routes1
Has abstractyes

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