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Record W2463966849 · doi:10.1016/j.jalz.2015.06.885

P3‐019: An Alzheimer's genetic risk composite, but not ApoE, intensifies diabetes‐related neurocognitive slowing in nondemented older adults

2015· article· en· W2463966849 on OpenAlexaff
G. Peggy McFall, Shraddha Sapkota, Sandra A. Wiebe, Kaarin J. Anstey, Roger A. Dixon

Bibliographic record

VenueAlzheimer s & Dementia · 2015
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicNutrition, Genetics, and Disease
Canadian institutionsUniversity of Alberta
Fundersnot available
KeywordsNeurocognitiveApolipoprotein EType 2 diabetesMedicinePsychologyCognitive declineDiseaseOncologyGerontologyInternal medicineDementiaDiabetes mellitusDemographyCognitionPsychiatryEndocrinology

Abstract

fetched live from OpenAlex

Risk factors (and their synergistic interactions) associated with Alzheimer's disease (AD) may predict normal or preclinical deficits and decline. Although ApoE (rs429358, rs7412) is the gene most consistently linked with AD risk, genome-wide association studies have identified others, including CLU (rs11136000), CR1 (rs6656401), and PICALM (rs541458). Type 2 diabetes (T2D) is a risk factor for AD and for increased cognitive deficits in nondemented older adults. We examined if the effect of diabetes on neurocognitive speed performance (level) and longitudinal change was intensified by (a) genetic risk from each of the four variants independently or (b) an AD Genetic Risk Composite (AGRC) representing combined risk from all four variants. This longitudinal design included non-demented older adults (n=591, baseline M age=69, age range 53–91, 68% women, 8% with T2D) followed over 9 years. Saliva was processed with standard procedures from Oragene-DNA Genotek. Genotyping was carried out using a PCR-RFLP strategy. The AGRC was created by summing allelic risk across the four specified genotypes: 0=no risk, 1=moderate risk, 2=full risk and then grouped into low and high risk using median split (Mdn=3.0). Statistical analyses included latent growth modeling testing independent and interactive effects on level (centering age=75) and change using a confirmed neurocognitive speed latent variable consisting of choice reaction time, sentence verification, and lexical decision measures. First, adults with T2D exhibited slower speed performance at age 75 than adults without T2D (b=.494, p=.007). Second, none of the genetic risk variants showed independent effects on speed performance or change. Third, interaction analyses (e.g., T2D x ApoE) showed no magnification of speed decrements. Fourth, intensification interaction analyses (T2D x AGRC) showed that adults in the high risk AGRC group with T2D exhibited significantly greater 9-year decline in speed (b=.048, p=.004). Finally, education and pulse pressure where significant covariates but did not alter the observed effects. Independently, as expected, diabetes was associated with cognitive slowing in nondemented aging. Only the AD Genetic Risk Composite, not ApoE or other variants, intensified the effects of T2D on neurocognitive speed in the form of exacerbated slowing over 9 years.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.008

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.017
GPT teacher head0.248
Teacher spread0.231 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations1
Published2015
Admission routes1
Has abstractyes

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