MétaCan
Menu
Back to cohort
Record W2477533041 · doi:10.1158/1538-7445.am2016-2179

Abstract 2179: Macrolide toxin Mycalolide B is a potent inhibitor of HER2 cancer cell invasion and is the basis of actin targeted therapy for metastatic cancers

2016· article· en· W2477533041 on OpenAlexaff
Rodette Williams, Andrew W. Craig, John S. Allingham

Bibliographic record

VenueCancer Research · 2016
Typearticle
Languageen
FieldMedicine
TopicCancer Treatment and Pharmacology
Canadian institutionsQueen's University
Fundersnot available
KeywordsSKBR3Cancer researchTrastuzumabAntibody-drug conjugateMetastasisCancer cellCancerMedicineBiologyBreast cancerAntibodyImmunologyInternal medicineMonoclonal antibody

Abstract

fetched live from OpenAlex

Abstract Breast and ovarian cancers are among the leading causes of cancer related deaths in women worldwide. Human epidermal growth factor receptor 2 (HER2) is highly expressed in a subset of these cancers that show high rates of progression to metastatic disease. Metastasis is driven by filamentous actin (F-actin) based protrusions that penetrate and degrade extracellular matrix (ECM) to facilitate tumor invasion. In this study, we tested an actin-depolymerizing macrolide toxin Mycalolide B, as a potential suppressor of highly metastatic HER2+ breast and ovarian cancer cell models. Dose responses with Mycalolide B in SKBR3 breast cancer and SKOV3 ovarian cancer cells resulted in similar cytotoxicity (∼65 nM IC50) profiles. In addition, Mycalolide B doses well below the IC50 showed potent suppression of leading edge protrusions, and motility in SKBR3 and SKOV3 cancer cells. This also correlated with reduced ECM degradation and Transwell invasion at low nanomolar doses of Mycalolide B. In contrast, other F-actin based processes such as endocytosis of HER2 and EGFR, were less sensitive to Mycalolide B treatment. However, Mycalolide B treatment did skew the size of endocytic vesicles, which may reflect defects in F-actin based vesicle motility or maturation. Given that HER2 cancers have been effectively targeted by Trastuzumab and Trastuzumab-based antibody-drug conjugates, we are currently testing the compatibility of combined treatments with Mycalolide B and Trastuzumab, for their effects on F-actin based invasion of HER2+ cancers. This strategy may yield a novel class of antibody-drug conjugate, and lead to new HER2 targeted therapies that suppress tumor metastasis by disrupting early steps that are dependent on efficient F-actin polymerization. Citation Format: Rodette N. Williams, Andrew W. Craig, John S. Allingham. Macrolide toxin Mycalolide B is a potent inhibitor of HER2 cancer cell invasion and is the basis of actin targeted therapy for metastatic cancers. [abstract]. In: Proceedings of the 107th Annual Meeting of the American Association for Cancer Research; 2016 Apr 16-20; New Orleans, LA. Philadelphia (PA): AACR; Cancer Res 2016;76(14 Suppl):Abstract nr 2179.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.005

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.111
GPT teacher head0.439
Teacher spread0.328 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2016
Admission routes1
Has abstractyes

Explore more

Same venueCancer ResearchSame topicCancer Treatment and PharmacologyFrench-language works237,207