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Record W2523361297 · doi:10.1002/ajmg.a.37940

Phenotype and genotype in 52 patients with Rubinstein–Taybi syndrome caused by <i>EP300</i> mutations

2016· article· en· W2523361297 on OpenAlexaff
Patricia Fergelot, Martine van Belzen, Julien Van‐Gils, Alexandra Afenjar, Christine M. Armour, Benoı̂t Arveiler, Lex Beets, Lydie Bürglen, Tiffany Busa, Marie Collet, Julie Deforges, Bert B.A. de Vries, Elena Garrido, Nathalie Dorison, Juliette Dupont, Christine Francannet, Sixto García‐Miñáur, Elisabeth Gabau Vila, Samuel Gebré‐Medhin, Blanca Gener Querol, David Geneviève, Marion Gérard, Cristina Gervasini, Alice Goldenberg, Dragana Josifova, Katherine Lachlan, Saskia M. Maas, Bruno Maranda, Jukka S. Moilanen, Ann Nordgren, P. Parent, Julia Rankin, William Reardon, Marlène Rio, J. Roume, Adam Shaw, Robert Śmigiel, A Sojo, Benjamin D. Solomon, Agnieszka Stembalska, Constance T. R. M. Stumpel, F J Ortega Suárez, Paulien A. Terhal, Simon Thomas, Renaud Touraine, Alain Verloès, Catherine Vincent‐Delorme, Josephine Wincent, Dorien J.M. Peters, Oliver Bartsch, Lidia Larizza, Didier Lacombe, Raoul C. M. Hennekam

Bibliographic record

VenueAmerican Journal of Medical Genetics Part A · 2016
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicCongenital limb and hand anomalies
Canadian institutionsChildren's Hospital of Eastern Ontario
Fundersnot available
KeywordsRubinstein–Taybi syndromeBiologyMissense mutationGeneticsMicrocephalyPhenotypeCREB-binding proteinMutationIntellectual disabilityGeneGenotypeTranscription factor

Abstract

fetched live from OpenAlex

Rubinstein-Taybi syndrome (RSTS) is a developmental disorder characterized by a typical face and distal limbs abnormalities, intellectual disability, and a vast number of other features. Two genes are known to cause RSTS, CREBBP in 60% and EP300 in 8-10% of clinically diagnosed cases. Both paralogs act in chromatin remodeling and encode for transcriptional co-activators interacting with >400 proteins. Up to now 26 individuals with an EP300 mutation have been published. Here, we describe the phenotype and genotype of 42 unpublished RSTS patients carrying EP300 mutations and intragenic deletions and offer an update on another 10 patients. We compare the data to 308 individuals with CREBBP mutations. We demonstrate that EP300 mutations cause a phenotype that typically resembles the classical RSTS phenotype due to CREBBP mutations to a great extent, although most facial signs are less marked with the exception of a low-hanging columella. The limb anomalies are more similar to those in CREBBP mutated individuals except for angulation of thumbs and halluces which is very uncommon in EP300 mutated individuals. The intellectual disability is variable but typically less marked whereas the microcephaly is more common. All types of mutations occur but truncating mutations and small rearrangements are most common (86%). Missense mutations in the HAT domain are associated with a classical RSTS phenotype but otherwise no genotype-phenotype correlation is detected. Pre-eclampsia occurs in 12/52 mothers of EP300 mutated individuals versus in 2/59 mothers of CREBBP mutated individuals, making pregnancy with an EP300 mutated fetus the strongest known predictor for pre-eclampsia. © 2016 Wiley Periodicals, Inc.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.511
Threshold uncertainty score0.309

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.004
GPT teacher head0.212
Teacher spread0.209 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations122
Published2016
Admission routes1
Has abstractyes

Explore more

Same venueAmerican Journal of Medical Genetics Part ASame topicCongenital limb and hand anomaliesFrench-language works237,207