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Clinicopathologic Subtypes of Mantle Cell Lymphoma (MCL) Show Distinct Patterns of Genetic Copy Number Alteration.

2006· article· en· W2563578391 on OpenAlexaff
Ronald J. deLeeuw, Chad A. Malloff, Lindsey R. Kimm, Joseph M. Connors, Martin J.S. Dyer, Randy D. Gascoyne, Wan L. Lam

Bibliographic record

VenueBlood · 2006
Typearticle
Languageen
FieldMedicine
TopicLymphoma Diagnosis and Treatment
Canadian institutionsBC Cancer AgencyCanada's Michael Smith Genome Sciences Centre
Fundersnot available
KeywordsBlastoidMantle cell lymphomaBiologyComparative genomic hybridizationGenome instabilityCancer researchLymphomaChromosomal translocationGeneticsGeneChromosomeImmunologyDNA damage

Abstract

fetched live from OpenAlex

Abstract Introduction: MCL is an aggressive NHL characterized by a t(11;14)(q13;q32). However this translocation alone cannot initiate lymphomagenesis and multiple secondary genomic alterations are necessary to induce the neoplastic phenotype. In addition, three distinct clinicopathologic subtypes of MCL (classic, blastoid, and leukemic) have been identified with differing prognosis. Secondary genomic alterations associated with each subtype have been poorly defined. Experimental Design: Genomic profiles from a panel of MCL samples comprised of 20 classic, 5 leukemic (no nodal involvement), and 12 blastoid cases, were created using a tiling-resolution BAC array CGH platform. Alignment of profiles to identify regions of recurrent alteration was accomplished using a custom developed software package (SeeGH). In addition, specific genes were analyzed for copy number status. Statistically significant distributions were determined using Chi square and Fisher’s exact analyses. Results: Twenty two regions of recurrent alteration were identified that were statistically different between subtypes (partial data in Table 1). As expected chromosome 9 losses were more prevalent in blastoid MCL. 13q34 losses were prevalent within classic and blastoid MCL, as opposed to leukemic MCL, which showed gains of this region. Analysis of biological pathways revealed that apoptotic signaling is not disrupted via copy number in a large portion of MCL cases (21%), while chromosomal instability genes show copy number disruption in 67% of MCL cases. Surprisingly, the glioblastoma pathway was preferentially disrupted in blastoid MCL (p=1.09x10−5), affecting all cases; whereas the p53 pathway was disrupted via copy number more often in leukemic MCL (p=0.0207). Conclusions: Multiple genomic regions and cancer related genes show a differential pattern of altered copy number distribution between MCL subtypes. These regions and genes likely contribute to the different clinicopathologic subtypes of MCL cases. Differential regions in MCL subtypes +/− chr. band size (Mb) clas% leuk% blast% enriched in deficient in − 1q25.2-q31.2 15.50 0 20 33 blast (p=0.03) class (p=0.01) − 2q26.3 2.91 5 0 33 blast (p=0.03) + 3cent-p12.1 4.18 0 60 25 leuk (p=0.02) class (p=0.005) + 7q11.23 3.24 0 0 33 blast (p=0.008) class (p=0.04) − 9p21.3 1.02 10 20 83 blast (p=0.00001) class (p=0.002) − 9q21.13 1.10 5 40 75 blast (p=0.0003) class (p=0.0002) − 9q21.2-q31.1 22.87 5 40 67 blast (p=0.001) class (p=0.0007) − 9q31.1 0.83 0 40 67 blast (p=0.0004) class (p=0.00006) − 9q34.12-qter 7.27 0 20 42 blast (p=0.009) class (p=0.005) + 10p12.2-p12.31 0.63 5 0 42 blast (p=0.009) − 13q34 1.69 40 0 75 blast (p=0.03) leuk (p=0.05) − 17p13.3-pter 2.28 5 60 25 leuk (p=0.04) − 17p13.1 1.21 5 60 25 leuk (p=0.04)

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.007

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.001
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.010
GPT teacher head0.247
Teacher spread0.236 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations1
Published2006
Admission routes1
Has abstractyes

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