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Record W2567618958 · doi:10.1016/s1525-0016(16)34108-9

499. Retinal Gene Therapy for Peroxisome Biogenesis Disorders

2015· article· en· W2567618958 on OpenAlexaff
Joseph G. Hacia, Jeannette Bennicelli, Ning Huang, Nancy Braverman, Steven J. Steinberg, Jean Bennett

Bibliographic record

VenueMolecular Therapy · 2015
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicPeroxisome Proliferator-Activated Receptors
Canadian institutionsMcGill University
Fundersnot available
KeywordsBiologyZellweger syndromePeroxisomal disorderRetinal degenerationRetinaRetinalPathologyGeneticsMedicineGeneNeurosciencePeroxisomeBiochemistry

Abstract

fetched live from OpenAlex

Peroxisome biogenesis disorders (PBDs) are a group of autosomal recessive disorders most frequently caused by inherited defects in the PEX1 gene, which is required for normal peroxisome assembly and biochemical functions. Approximately 80% of patients fall within the category of Zellweger spectrum disorder (PBD-ZSD), which has an overall incidence of 1:50,000 births in the United States. The majority of patients have milder forms of disease compatible with survival through adulthood; however, they typically show mild to moderate developmental delays, and progressive vision and hearing loss. Thus, therapeutic interventions that prevent or slow down visual loss could have a profound impact on the lives of these individuals Here, we present a retinal gene therapy approach that addresses visual deterioration in patients with milder forms of disease. Optical coherence tomography (OCT) has demonstrated that the cone photoreceptor cells are most significantly affected by loss of peroxisome functions in such patients. These visual phenotypes are recapitulated in a new mouse model of disease that expresses the murine equivalent of most common PEX1 mutation found in patients (PEX1-p.G843D). Electroretinogram (ERG) analyses indicated severe impairment of the cone visual pathway in these homozygous Pex1-mutant mice by 4 months of age with the rod visual system being relatively preserved. Staining retinal sections with peanut agglutinin showed that some cone photoreceptors were retained in the homozygote murine Pex1 -mutant retina at 3 weeks, but were completely degenerated in the adult. Finally, genome-wide expression studies showed specific reduction of photoreceptor cone-specific genes with no differential expression of other cell lineage-specific genes or others that indicate cell death or stress responses. We have developed AAV vectors to deliver normal copies of the 3.85-kb human PEX1 gene to the mammalian retina. These vectors use the rhodopsin kinase 1 (RK1) or truncated cytomegalovirus (CMV) promoters, respectively. Subretinal injections of RK1.PEX1. rAAV9 in healthy mice resulted in robust expression of human PEX1 mRNA. Furthermore, transduction of cultured Pex1 -mutant murine skin fibroblasts with CMV.PEX1.rAAV9 results in the rescue of peroxisome assembly defects in these cells, as determine by the intracellular localization of a modified GFP reporter protein with a peroxisome targeting signal. This indicates that the human PEX1 transgene can complement the genetic defect in the murine Pex1 gene. We are currently testing the ability of the AAV9-PEX1 gene delivery system to complement the retinal gene defect in our Pex1 -mutant mouse model. If successful, this could provide the preliminary data required to begin initial planning for eventual clinical trials in patients.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesMeta-epidemiology (narrow)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.176
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.020
GPT teacher head0.266
Teacher spread0.246 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations1
Published2015
Admission routes1
Has abstractyes

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