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Record W2587227370 · doi:10.1093/ecco-jcc/jjx002.875

P754 X-linked inhibitor of apoptosis protein genetic variants in paediatric-onset inflammatory bowel disease

2017· article· en· W2587227370 on OpenAlexaff
Eileen Crowley, Abdul Elkadri, Neil Warner, S. Zhang, Julie Horowitz, Jeffrey Staples, John D. Overton, Frederick E. Dewey, Claudia Gonzaga‐Jauregui, A M Griffiths, Aleixo M. Muise

Bibliographic record

VenueJournal of Crohn s and Colitis · 2017
Typearticle
Languageen
FieldMedicine
TopicAutoimmune and Inflammatory Disorders Research
Canadian institutionsSickKids FoundationUniversity of TorontoHospital for Sick Children
Fundersnot available
KeywordsInflammatory bowel diseaseMedicineXIAPSanger sequencingCrohn's diseaseUlcerative colitisDiseaseInhibitor of apoptosisExome sequencingImmunologyHemophagocytic lymphohistiocytosisPhenotypeGastroenterologyGeneticsMutationInternal medicineGeneBiologyApoptosis

Abstract

fetched live from OpenAlex

Background: Inflammatory bowel disease (IBD) has a multifactorial aetiology, with complex interactions between genetic and environmental factors. Recent studies have suggested an increasing spectrum of human monogenic diseases that can present with IBD-like intestinal inflammation. Mutations in X-linked inhibitor of apoptosis protein (XIAP) result in an X-linked recessive disorder whose phenotype is highly heterogeneous with respect to age at presentation and severity of disease and is poorly understood. Due to X chromosome inactivation heterozygous female carriers can also be symptomatic. Although a severe phenotype with infantile onset disease and predisposition to hemophagocytic lymphohistiocytosis (HLH) or X-linked lymphoproliferative syndrome (XLP) is recognized, one recent study has reported XIAP variants in 4% of male pediatric IBD patients. Methods: 1250 individual Hospital for Sick Children pediatric IBD patients have undergone whole exome sequencing in collaboration with the Regeneron Genetics Center. Within the cohort, 39 variants were called across the XIAP gene – 29 of which were high quality, rare (maf <0.01), protein coding variants predicted to be deleterious. 13 of these variants were present in 15 affected patients all of which have been Sanger validated. Case notes were retrospectively reviewed to ascertain phenotypic features of IBD in these 15 pediatric patients. Results: Of the 15 patients (80% males), 14 (93%) were diagnosed with IBD. 13 (92%) of these with Crohn's disease and 1 (7%) with ulcerative colitis. At the time of diagnosis, CD involved small bowel and colon (L3) in 50% children, colon (L2) in 14% and ileum (L1) in 28%. 21% of the children had perianal disease, with a further 21% having extra intestinal manifestations of IBD (erythema nodosum, fevers, large joint arthritis). A further infant was diagnosed with primary HLH and received a bone marrow transplant. This infant did have some gastrointestinal involvement with diffuse damage noted in the colonic mucosa and frequent apoptotic cells. 14% of these children had a first degree relative with IBD. 21% underwent ileocaecal resection with 57% progressing to biologic therapy. Patient samples, monocytes, are now undergoing a functional test to determine tumour necrosis factor (TNF) production of monocytes in response to NOD2 stimulation by muramyl dipeptides (L18-MDP) for the functional diagnosis of XIAP deficiency. Conclusions: Analysis of this large pediatric cohort confirms the highly varied phenotypic spectrum of IBD associated with XIAP mutations. Functional studies may more completely explain the observed variation.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.002
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.008

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.002
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.001
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.013
GPT teacher head0.273
Teacher spread0.260 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2017
Admission routes1
Has abstractyes

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