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Ten Days of Free Wheel Running Alters Cortical Glutamate‐Linked Mitochondrial Respiration in Rat Brain

2016· article· en· W2596652688 on OpenAlex
M. Anderson George, Karen Brebner, Daniel A. Kane

Why this work is in the frame

A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.

affAt least one author lists a Canadian institution in the pinned OpenAlex snapshot.
aboutThe title or abstract carries a Canadian signal from the geographic lexicon.

Bibliographic record

VenueThe FASEB Journal · 2016
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicGenetics and Physical Performance
Canadian institutionsSt. Francis Xavier University
Fundersnot available
KeywordsRespirationGlutamate receptorWheel runningChemistryNeuroscienceBiologyEndocrinologyAnatomyBiochemistryReceptor

Abstract

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The mechanisms through which exercise benefits brain function are not completely understood. Appreciation for the role of lactate as a major neuroenergetic fuel, particularly during exercise, has progressed in recent decades; however, the mitochondrial involvement in brain lactate metabolism is not clear. To explore the mechanisms involved, we examined mitochondrial lactate oxidation in brain and skeletal muscle from exercised rats. Respiratory oxygen flux ( J O 2 ) was measured in saponin‐permeabilized prefrontal cortex (PFC) and hippocampus (dentate gyrus; DG) samples, as well as red and white gastrocnemius muscle samples from five male Wistar rats (age: approximately 104 days) given access to running wheels. Over ten days of free wheel running, rats attained a maximal daily running distance averaging 2.1 ± 0.5 km. The wheel‐running rats remained relatively weight stable, losing 1.4 ± 3.1 g body mass on average, whereas the five paired control rats gained an average of 39.4 ± 2.8 g over the ten days. On day eleven, high resolution respirometry was performed on exercise and control samples ex vivo . The addition of lactate failed to affect mitochondrial J O 2 (ADP + malate) from either brain or muscle in either group. However, J O 2 rose significantly following the addition of exogenous NAD + in each tissue ( P < 0.01), implying the lactate dehydrogenase reaction operates outside of the mitochondrial matrix. This interpretation was further supported by inhibition of J O 2 following addition of an inhibitor of pyruvate transport, UK‐5099 ( P < 0.01). Interestingly, the subsequent addition of glutamate resulted in a disparate increase in mitochondrial J O 2 among tissues and treatment. Compared to controls, glutamate‐supported mitochondrial J O 2 in wheel runners was significantly greater in PFC ( P < 0.05), but not DG. This rise in J O 2 likely reflects activation of the malate‐aspartate shuttle through elevated mitochondrial glutamate transport. Paradoxically, wheel runners exhibited lower glutamate‐supported J O 2 in red gastrocnemius ( P < 0.05). This may be due to the influence of regular exercise on the putative lactate‐malate‐aspartate shuttle. The lactate‐malate‐aspartate shuttle is the interaction between the lactate and malate‐aspartate shuttles to translocate reducing power to the mitochondria. The decline in lactate dehydrogenase activity in skeletal muscle that accompanies aerobic exercise training is expected to negatively influence lactate‐malate‐aspartate shuttling, and may explain our observations in muscle. Collectively, the results of this study suggest that electron shuttling in rat brain following ten days of voluntary exercise is upregulated in PFC. These results also provide further evidence for an indirect mechanism of mitochondrial lactate oxidation in both brain and skeletal muscle. Support or Funding Information The Natural Sciences and Engineering Research Council of Canada; The Nova Scotia Health Research Foundation

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Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.008
Threshold uncertainty score0.215

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.013
GPT teacher head0.246
Teacher spread0.233 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it