MétaCan
Menu
Back to cohort
Record W2598285911 · doi:10.1093/schbul/sbx021.243

176.1 Bioenergetic Uncoupling of Astrocytes and Neurons May Underlie Cognitive Deficits in Schizophrenia

2017· article· en· W2598285911 on OpenAlexaff
Courtney R. Sullivan, Sinead M. O’Donovan, Amy J. Ramsey, Robert E. McCullumsmith

Bibliographic record

VenueSchizophrenia Bulletin · 2017
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicAmino Acid Enzymes and Metabolism
Canadian institutionsUniversity of Toronto
Fundersnot available
KeywordsGlutamatergicNeuroscienceBiologyPrefrontal cortexSchizophrenia (object-oriented programming)NeurotransmissionBiochemistryGlutamate receptorMedicineCognition

Abstract

fetched live from OpenAlex

Background: Pharmacologic, genetic, and theoretical considerations have firmly established schizophrenia as a disorder of excitatory synapses. Converging evidence from human schizophrenia studies and animal models of “broken” synapses suggest developing a brain with defective glutamatergic synapses yields metabolic abnormalities. This may be due to failure of astrocytes to develop/maintain metabolic coupling with glutamatergic neurons. Neurotransmission relies heavily on this metabolic coupling. For example, glycogen-rich glial cells synthesize pyruvate from glucose, convert pyruvate to lactate via lactate dehydrogenase (LDH), and transport lactate via monocarboxylate transporters (MCTs) to neurons for energetic use (the “lactate shuttle”). Working memory performance and long-term potentiation in rodents is impaired following disruption of MCTs. These findings suggest that bioenergetic coupling via the lactate shuttle is tightly coupled to cognitive function; thus, we hypothesized that these pathways are important pathophysiological substrates in chronic schizophrenia. Methods: To test this hypothesis, we assessed the integrity of the lactate shuttle and related metabolic pathways by adapting commercially available assays for use with postmortem brain. Results: We found significant decreases in LDH (~10%, P = .0484) and hexokinase (HXK; 24%, P = .0024) enzyme activity in the dorsolateral prefrontal cortex in schizophrenia. LDH and HXK activity were unchanged in the frontal cortex of haloperidol treated rats. There were no region-level changes in LDH, LDHA, LDHB, HXK, or MCT1 protein levels in schizophrenia. However, at the cell level, we found increases in MCT1 mRNA expression in both astrocytes and neurons. We have also assessed expression of metabolic substrates in an animal model of schizophrenia, the NR1 knockdown mouse. In this model of “broken” excitatory synapses, we found a 63% decrease in astrocytic MCT4 and 60% decrease in neuronal GLUT3 transcripts in NR1 KD mice versus wildtype. Conclusion: Our data suggest that LDH and HXK enzyme activity, and not expression, is decreased in schizophrenia, diminishing the ability of astrocytes to supply neurons with energetic substrates, which may impact cognition. Our animal studies support this hypothesis and suggest that alterations in the lactate shuttle and related pathways and metabolites in schizophrenia may be high-yield substrates to target for future drug development or imaging-based biomarkers. This work was supported by the NIMH (MH09445).

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesMeta-epidemiology (narrow)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.324
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.013
GPT teacher head0.251
Teacher spread0.238 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations2
Published2017
Admission routes1
Has abstractyes

Explore more

Same venueSchizophrenia BulletinSame topicAmino Acid Enzymes and MetabolismFrench-language works237,207