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Record W2621944282

Bazooka and atypical Protein Kinase C regulate dynamic actomyosin networks during Drosophila amnioserosa apical constriction

2013· dissertation· en· W2621944282 on OpenAlexfundno aff
Daryl J. V. David

Bibliographic record

VenueTSpace (University of Toronto) · 2013
Typedissertation
Languageen
FieldNeuroscience
TopicNeurobiology and Insect Physiology Research
Canadian institutionsnot available
FundersNatural Sciences and Engineering Research Council of CanadaUniversity of Toronto
KeywordsDrosophila (subgenus)Apical constrictionCell biologyBiologyChemistryBiochemistryMorphogenesisGene
DOInot available

Abstract

fetched live from OpenAlex

Cell shape changes drive tissue remodeling. Polarized activity of actin and myosin drive apical constriction, as seen for example in Drosophila embryonic amnioserosa cells during dorsal closure (DC). My first aim was to characterize interactions between apical actomyosin networks and the cell polarity regulators Bazooka (Baz, the Drosophila Par-3), Par-6, and atypical Protein Kinase C (aPKC) (the PAR complex) during Drosophila DC. I found that both actomyosin networks and the PAR complex are enriched at the apical surfaces of amnioserosa cells and that actomyosin contractility is driven by cyclical assembly and disassembly of actomyosin networks. The pulsatile actomyosin networks translocate across persistent apical surface PAR complex puncta. To assess whether the PAR complex interacts with actomyosin, I characterized myosin dynamics with PAR loss- and gain-of-function perturbations. Baz enhances, whereas Par6/aPKC inhibits actomyosin. These studies suggest that PAR proteins regulate pulsatile apical actomyosin networks mediating constriction of amnioserosa cells. My next aim was to characterize actomyosin networks during a shift in their dynamics. Amnioserosa constriction transitions from pulsatile to persistent from early to late DC. Since oscillatory networks result from delayed negative feedback, I examined whether such regulation exists in these actomyosin networks. The actomyosin inhibitor aPKC is recruited to the apical surface by actomyosin and, in turn, aPKC recruits Baz. To examine the significance of Baz – aPKC dynamic interactions, I ectopically stabilized their interactions. This served to inhibit the antagonism of actomyosin networks by aPKC, suggesting that Baz can act as a competitive inhibitor of aPKC. I found that interactions between Par-6 and Baz increase during DC, suggesting an increase in aPKC inhibition during DC progression. To examine whether decreased inhibition can tune oscillatory actomyosin networks, we collaborated with Qiming Wang and Dr. James Feng (University of British Columbia) to test this oscillation in silico. Computer modeling suggests that decreasing delayed negative feedback can transition actomyosin oscillations towards stabilized constriction. Together, these results demonstrate the requirement of Baz – aPKC interactions for their localization and for dynamic regulation of aPKC by a competitive inhibitor. Finally, my research reveals a regulatory circuit to tune actomyosin behaviour during development.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.003

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.011
GPT teacher head0.247
Teacher spread0.236 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2013
Admission routes1
Has abstractyes

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