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Analysis of BRCA1-related functional associations in sporadic triple negative breast cancer: A network-based approach.

2016· article· en· W2710457918 on OpenAlexaff
Arcangela De Nicolo, Benjamin Haibe‐Kains, Murat Taşan, Michael E. Cusick, Marc Vidal, John Quackenbush, Vladimir Joukov, David M. Livingston

Bibliographic record

VenueJournal of Clinical Oncology · 2016
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicBioinformatics and Genomic Networks
Canadian institutionsPrincess Margaret Cancer CentreUniversity of TorontoUniversity Health Network
Fundersnot available
KeywordsTriple-negative breast cancerBreast cancerTranscriptomeMedicineComputational biologyCancer researchBioinformaticsCancerGeneBiologyGeneticsGene expressionInternal medicine

Abstract

fetched live from OpenAlex

1070 Background: Sporadic triple negative breast cancers (TNBC) are aggressive malignancies that present a yet unaddressed clinical challenge. They are refractory to hormonal and HER2-targeted therapy, leaving chemotherapy as the mainstay of systemic treatment. Their striking resemblance to BRCA1-mutated hereditary breast cancers has led to the speculation that BRCA1 pathway derangement underlies sporadic TNBC and could be exploited therapeutically. BRCA1 has been implicated in multiple cellular processes via interactions with diverse partner proteins. Methods: To search for TNBC-specific aberrations within the complex array of BRCA1-related functional associations, we devised a strategy that takes into account the versatility of the BRCA1 protein and combines database mining, literature curation, network modeling, and transcriptome analysis. Results: We used database- and literature-derived data on protein-protein interactions, co-complex memberships, and biochemical modifications involving BRCA1 to build a BRCA1-centered Interaction network of 157 known BRCA1 partners. We applied functional linkage analysis to add predicted functional links and generate a highly-connected Shell network of 1137 nodes and 2941 edges, directly or indirectly associated with BRCA1. We next used a curated compendium of 14 datasets comprising 2022 uniformly subtyped sporadic primary breast cancers to compute, meta-analytically, the relative expression of the network components in TNBC vs non-TNBC subtypes. Lastly, we derived TNBC-specific subnetworks of significantly hyperactive or suppressed BRCA1-functionally associated genes. Conclusions: Via network analysis, we have identified presumptive signatures of BRCA1 pathway deregulation in sporadic TNBC. We will assess their prognostic/predictive value and potential use for patient stratification. Where relevant, we will also employ them as hypothesis-generating tools to prioritize BRCA1-related genes/proteins and pathways for in-depth analyses and to search for therapeutically exploitable vulnerabilities. These studies can help to inform therapy selection and tailored treatment and, hence, guide clinical trial design.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.003
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.006
Threshold uncertainty score0.013

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.003
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0000.001
Bibliometrics0.0060.004
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0010.001
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.042
GPT teacher head0.351
Teacher spread0.310 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2016
Admission routes1
Has abstractyes

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