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Genome-wide association study (GWAS) of cisplatin-related hearing loss in testicular cancer survivors (TCS) to reveal associated variant in Wolfram syndrome 1 (WFS1) gene.

2016· article· en· W2730272516 on OpenAlexaff
Heather E. Wheeler, Robert D. Frisina, Eric R. Gamazon, Omar El Charif, Darren R. Feldman, Robert J. Hamilton, David J. Vaughn, Clair J. Beard, Chunkit Fung, Lawrence H. Einhorn, Taisei Mushiroda, Michiaki Kubo, Nancy J. Cox, M. Eileen Dolan, Lois B. Travis

Bibliographic record

VenueJournal of Clinical Oncology · 2016
Typearticle
Languageen
FieldAgricultural and Biological Sciences
TopicPlant Molecular Biology Research
Canadian institutionsPrincess Margaret Cancer CentreUniversity Health Network
Fundersnot available
KeywordsGenome-wide association studySingle-nucleotide polymorphismMinor allele frequencyHearing lossWolfram syndromeMedicine1000 Genomes ProjectGeneticsSNPGenetic associationExpression quantitative trait lociOncologyGenotypeDiabetes mellitusBiologyEndocrinologyAudiologyGene

Abstract

fetched live from OpenAlex

10015 Background: Cisplatin is widely used and highly ototoxic; we sought to identify genetic variants that modulate cisplatin-related hearing loss. Methods: We performed a GWAS for cisplatin-related hearing loss in 512 patients of European genetic ancestry enrolled in an ongoing multi-center North American clinical study of TCS. The geometric mean of air conduction thresholds measured at 4, 6, 8, 10 and 12 kHz was the quantitative phenotype used in the GWAS. SNPs were genotyped on the Illumina HumanOmniExpressExome chip and imputed using the 1000 Genomes reference panel, with 5.1 million SNPs passing quality control for GWAS inclusion. Covariates included in the GWAS were age at audiometry, cumulative cisplatin dose, and 10 genotypic principal components. Results: One SNP, rs62283056, in the first intron of WFS1 met genome-wide significance for association with cisplatin-associated hearing loss (P = 4.8 x 10-9). Mutations in this gene can cause autosomal dominant deafness and Wolfram syndrome, also called DIDMOAD (Diabetes Insipidus, Diabetes Mellitus, Optic Atrophy, and Deafness). The minor allele of rs62283056 (frequency 0.21 in the GWAS cohort) associates with increased hearing loss and decreased expression of WFS1 (cis-eQTL) in several human tissues from the GTEx Project, including cerebellar hemisphere, tibial nerve and thyroid. WFS1 is differentially expressed in the human inner ear (cochlea vs. vestibule, Padj = 0.032, Schrauwen et al. Hear Res 2015). We found a significant interaction between rs62283056 genotype and cumulative cisplatin dose (P = 0.029), indicating higher dose may exacerbate the hearing loss effect in patients carrying the minor allele. When hearing loss is dichotomized as a geometric mean > 20 dB (n = 242 affected), the allelic OR for hearing loss risk is 1.9 [95% CI: 1.3 – 2.8]. Conclusions: The WFS1 SNP is the first report of a genome-wide significant association with cisplatin-related hearing loss in adults. Expression data and known gene function corroborate its potential involvement in hearing loss. Future studies will focus on replicating and extending the genetic and mechanistic findings discovered here.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.005
Threshold uncertainty score0.011

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.001
Bibliometrics0.0010.001
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0030.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.082
GPT teacher head0.387
Teacher spread0.305 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations1
Published2016
Admission routes1
Has abstractyes

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