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Record W2741419410 · doi:10.1158/1538-7445.am2017-3247

Abstract 3247: Discovery of a novel drug that affects centrosome clustering

2017· article· en· W2741419410 on OpenAlexaff
Dilan B. Jaunky, Kevin Larocque, Javier Porro Suardiaz, Dan Yang, Emma J. Furze, Pat Forgione

Bibliographic record

VenueCancer Research · 2017
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicMicrotubule and mitosis dynamics
Canadian institutionsConcordia University
Fundersnot available
KeywordsCentrosomeNocodazoleMitosisMicrotubuleCancer cellBiologyCell biologyCancerCancer researchPaclitaxelCellCell cycleBiochemistryGeneticsCytoskeleton

Abstract

fetched live from OpenAlex

Abstract Our goal is to identify molecular regulators of a mechanism that occurs uniquely in cancer cells, and to develop a selective anti-cancer drug. Most chemotherapies are non-selective, causing severe side-effects. In additions, cancers often develop resistance to some of the more commonly used chemotherapies. To expand the repertoire of available drugs, and to design drugs that are selective, we need to identify molecules that regulate the physiological changes that occur primarily in cancer cells. For example, cancer cells in many hard-to-treat cancers have aberrant centrosomes, which may be supernumerary or fragmented. During mitosis, these aberrant centrosomes must cluster to form bipolar spindles for successful division. Thus, targeting a process like centrosome clustering is ideal, since it is not necessary in healthy cells. We synthesized a small, stable scaffold with amenability to structure-activity-relationship studies, and found several analogues with IC50 values < 50 nM, depending on the cancer cell line. Preliminary tests showed that these compounds prevent tumors from forming and/or cause their regression in vitro. We performed cell biological studies to characterize their mechanism of action. In several different types of cancer cells, these compounds cause mitotic arrest and centrosome declustering at concentrations where they have little affect on non-cancerous cells. Live imaging revealed that within minutes of adding the compounds to HeLa cells expressing GFP-tagged tubulin, we observed rapid microtubule depolymerization and centrosome declustering. After washing out the compound, microtubule polymerization recovered, but the mitotic spindles were multipolar. Adding similar concentrations of Nocodazole, a microtubule-depolymerizing drug, also caused rapid microtubule depolymerization, but after washing out the drug, the spindles were bipolar. We are in the process of identifying the molecular target of these compounds to provide crucial insight to the mechanism governing centrosome clustering, and are continuing to perform SAR studies to obtain compounds with higher efficacy and selectivity. Citation Format: Dilan B. Jaunky, Kevin Larocque, Javier Porro Suardiaz, Dan Yang, Emma J. Furze, Pat Forgione, Alisa Piekny. Discovery of a novel drug that affects centrosome clustering [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2017; 2017 Apr 1-5; Washington, DC. Philadelphia (PA): AACR; Cancer Res 2017;77(13 Suppl):Abstract nr 3247. doi:10.1158/1538-7445.AM2017-3247

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.010

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0030.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.057
GPT teacher head0.377
Teacher spread0.320 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2017
Admission routes1
Has abstractyes

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