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Record W2752715789 · doi:10.1161/str.47.suppl_1.tmp52

Abstract TMP52: Inactivated Microglia and Inhibition Neutrophil Infiltration Reduce Brain Injury Following Intracerebral Hemorrhage in Mice

2016· article· en· W2752715789 on OpenAlexaff
Yongmei Sun, Wenwu Chen, Min Liu, Yuming Xu, Yanhong Li, Jue He, Xiaohui Li, Fang Yuan, Lingxiu Jiang, Jian Fang, V. Wee Yong, Mengzhou Xue

Bibliographic record

VenueStroke · 2016
Typearticle
Languageen
FieldMedicine
TopicIntracerebral and Subarachnoid Hemorrhage Research
Canadian institutionsUniversity of Calgary
Fundersnot available
KeywordsMicrogliaMedicineIntegrin alpha MGenetically modified mouseInfiltration (HVAC)Intracerebral hemorrhagePathologyImmunologyGanciclovirPharmacologyTransgeneInflammationInternal medicineImmune systemBiologyBiochemistrySubarachnoid hemorrhageVirus

Abstract

fetched live from OpenAlex

Introduction: Microglia become activated and monocytes become macrophages following intracerebral hemorrhage (ICH). They are often called “microglia/macrophage” since it is difficult to differentiate between them. The neutrophils also infiltrate into the injured brain after ICH. Those cells are capable of releasing numerous detrimental mediators. Hypothesis: The activation of microglia/macrophage and the infiltrated neutrophils promote brain injury after ICH and that inhibition both of them attenuates brain injury following ICH. Methods: 10-μl of autologous blood obtained from tail was introduced into the right striatum of adult male mice to produce ICH injury. We used transgenic CD11b thymidine kinase (CD11b-TK) mice where proliferating microglia/macrophage were removed by ganciclovir treatment. Functional grade purified anti-mouse Ly6G/Gr-1 antibodies were used to reduce the neutrophil infiltration. Groups of CD11b-TK mice were treated with ganciclovir and/or anti-mouse Ly6G/Gr-1 antibodies. These mice were killed at 3 days after ICH injury and brain sections were stained for various parameters. Results: ICH injury resulted in activation of microglia/macrophages through 1-7 days of ICH insult. The area of brain damage peaked at 2-3 days; the number of dying neurons peaked at 1-3 days, the number of infiltrated neutrophils peaked at 1-3 days, while the activation of microglia/macrophages peaked at days 3-4. These findings led us to choose day 3 for further studies. The activation of microglia/macrophage was significantly reduced in CD11b-TK transgenic mice with ganciclovir treatment after ICH. The numbers of infiltrated neutrophils were significantly reduced with anti-mouse Ly6G/Gr-1 antibodies treatment after ICH. Correspondingly, the area of brain damage and the extent of neuronal death were minimal in CD11b-TK mice with ganciclovir and anti-LyG6/Gr-1 antibodies treatment after ICH. Conclusions: Both activated microglia/macrophages and the infiltrated neutrophils after ICH promote brain injury. Thus inhibition of their activities reduces ICH brain injury. These results shed light on the advent of new medications for ICH patients, including microglia deactivators and the antibodies to alleviate neutrophil infiltration.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.010

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0010.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0030.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.014
GPT teacher head0.283
Teacher spread0.269 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2016
Admission routes1
Has abstractyes

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