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Brain Regions Showing White Matter Loss in Huntington’s Disease Are Enriched for Synaptic and Metabolic Genes

2017· article· en· W2765263555 on OpenAlexaff
Peter McColgan, Sarah Gregory, Kiran Seunarine, Adeel Razi, Marina Papoutsi, Eileanoir B. Johnson, Alexandra Dürr, Raymund A.C. Roos, Blair R. Leavitt, Peter Holmans, Rachael I. Scahill, Chris A. Clark, Geraint Rees, Sarah J. Tabrizi, Allison Coleman, Joji Decolongon, Ming Fan, Terri L. Petkau, C Jauffret, Damián Justo, Stéphane Lehéricy, K Nigaud, Romain Valabrègue, Anne Schoonderbeek, Ellen P. Hart, Davina J. Hensman Moss, Rajarshi Ghosh, Helen Crawford, C. Berna, D. Mahaleskshmi, Ralf Reilmann, N Weber, Izelle Labuschagne, Julie C. Stout, G. Bernhard Landwehrmeyer, Michael Orth, Isabella Mayer, Hans J. Johnson, D. Crawfurd

Bibliographic record

VenueBiological Psychiatry · 2017
Typearticle
Languageen
FieldNeuroscience
TopicGenetic Neurodegenerative Diseases
Canadian institutionsUniversity of British Columbia
FundersUniversität UlmNED University of Engineering and TechnologyMonash UniversityInstitut National de la Santé et de la Recherche MédicaleMedical Research CouncilCardiff UniversityUniversity College London Hospitals NHS Foundation TrustRosetrees TrustCentre National de la Recherche ScientifiqueCHDI FoundationUniversity of Engineering and Technology, LahoreWellcome TrustNational Institute for Health and Care ResearchNIHR Biomedical Research Centre, Royal Marsden NHS Foundation Trust/Institute of Cancer ResearchUniversity College LondonAssistance publique-Hôpitaux de ParisUniversiteit LeidenPan-Massachusetts Challenge
KeywordsWhite matterHuntington's diseaseNeuroscienceGeneDiseaseBiologyGeneticsMedicineInternal medicineMagnetic resonance imaging

Abstract

fetched live from OpenAlex

BACKGROUND: The earliest white matter changes in Huntington's disease are seen before disease onset in the premanifest stage around the striatum, within the corpus callosum, and in posterior white matter tracts. While experimental evidence suggests that these changes may be related to abnormal gene transcription, we lack an understanding of the biological processes driving this regional vulnerability. METHODS: Here, we investigate the relationship between regional transcription in the healthy brain, using the Allen Institute for Brain Science transcriptome atlas, and regional white matter connectivity loss at three time points over 24 months in subjects with premanifest Huntington's disease relative to control participants. The baseline cohort included 72 premanifest Huntington's disease participants and 85 healthy control participants. RESULTS: We show that loss of corticostriatal, interhemispheric, and intrahemispheric white matter connections at baseline and over 24 months in premanifest Huntington's disease is associated with gene expression profiles enriched for synaptic genes and metabolic genes. Corticostriatal gene expression profiles are predominately associated with motor, parietal, and occipital regions, while interhemispheric expression profiles are associated with frontotemporal regions. We also show that genes with known abnormal transcription in human Huntington's disease and animal models are overrepresented in synaptic gene expression profiles, but not in metabolic gene expression profiles. CONCLUSIONS: These findings suggest a dual mechanism of white matter vulnerability in Huntington's disease, in which abnormal transcription of synaptic genes and metabolic disturbance not related to transcription may drive white matter loss.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.030
Threshold uncertainty score0.730

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0010.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.060
GPT teacher head0.305
Teacher spread0.245 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations104
Published2017
Admission routes1
Has abstractyes

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