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Record W2767717304

Potential molecular mechanisms of insulin like growth factor-1 (IGF-1)/insulin like growth factor-1 receptor (IGF-1R) interaction in prostate cancer invasion

2004· article· en· W2767717304 on OpenAlexaff
Hemani C. Setya, Aline Fiebig‐Comyn, Gurmit Singh, Sujata Persad

Bibliographic record

VenueCancer Research · 2004
Typearticle
Languageen
FieldMedicine
TopicGrowth Hormone and Insulin-like Growth Factors
Canadian institutionsMcMaster UniversityJuravinski Cancer Centre
Fundersnot available
KeywordsDU145WortmanninInsulin-like growth factorMAPK/ERK pathwayGrowth factorCancer researchBiologyProstate cancerEndocrinologySignal transductionInternal medicineCell biologyCancerReceptorPI3K/AKT/mTOR pathwayMedicineLNCaP
DOInot available

Abstract

fetched live from OpenAlex

5030 Epidemiological studies suggest that elevated serum levels of the peptide growth factor, IGF-1 is linked to an increased risk of developing prostate cancer. However, little is known about a direct role of IGF-1/IGF-1R axis in prostate cancer progression to an invasive phenotype. The objective of this study is to investigate the role of IGF-1/IGF-1R in regulating the invasive phenotype of prostate cancer and evaluate the molecular mechanism/s involved in promoting this invasive potential. Binding of IGF-1 to its receptor IGF-1R is known to signal intracellularly via the phosphatidylinositol 3-kinase (PI3-K) pathway and the p42/44 mitogen activated protein kinase (MAPK) pathway. Invasion assays using Matrigel invasion chambers indicate that treatment of DU145 human prostate carcinoma cells with IGF-1 results in an increase in invasive potential of these cells and this stimulation is attenuated in the presence of the PI3-K inhibitor Wortmannin (W) and with the MAPK inhibitor PD98059 (PD). IGF-1 stimulation is also inhibited in the presence of an IGF-1R neutralizing antibody indicating the invasive phenotype of DU145 cells is due specifically to IGF-1 stimulation. The activities of the PI3-K and MAPK pathways, as determined by phosphorylation of protein kinase B and p42/44 MAP kinase respectively, indicated that both pathways are activated in the presence of IGF-1 and this stimulatory effect is inhibited in the presence of their inhibitors W and PD respectively. IGF-1/IGF-1R mediated regulation of the expression of matrix metalloproteinases (MMPs) was evaluated as a mechanism involved in promoting the invasive phenotype. Gelatin zymography identified that the activities of MMP-2 and –9 (two gelatinases highly associated with prostate cancer invasion) were stimulated by IGF-1 while MMP-1 activity remained unaltered. IGF-1 activation of MMP-2 and –9 was inhibited in the presence of W and PD. Western blot analysis of MMP-2 and MMP-9 protein expression showed that while MMP-9 expression paralleled the zymogram results, there was no change in MMP-2 expression. This indicates that while IGF-1 regulation of MMP-9 activity may be at the transcriptional level, MMP-2 is likely regulated by other mechanism/s. These results suggest that IGF-1 is involved in promoting prostate cancer invasion by regulating MMP-2 and 9 expression and activities through the PI3-K and MAPK pathways. (This research was supported by funds from the Hamilton Health Science research foundation).

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.011

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.001
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0010.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0030.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.041
GPT teacher head0.340
Teacher spread0.298 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations2
Published2004
Admission routes1
Has abstractyes

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