MétaCan
Menu
Back to cohort
Record W2767903200 · doi:10.1093/neuonc/nox168.1050

TMOD-11. HUMAN STEM CELL BASED MODEL OF MEDULLOBLASTOMA

2017· article· en· W2767903200 on OpenAlexaff
Miller Huang, Jignesh Tailor, Qiqi Zhen, Emily K. Nash, Joanna Phillips, A. Sorana Morrissy, Marcel Kool, Stefan M. Pfister, Fredrik J. Swartling, Michael D. Taylor, Austin Smith, William A. Weiss

Bibliographic record

VenueNeuro-Oncology · 2017
Typearticle
Languageen
FieldMedicine
TopicCancer Cells and Metastasis
Canadian institutionsHospital for Sick ChildrenUniversity of Toronto
Fundersnot available
KeywordsMedulloblastomaStem cellBiologyCell biologyCancer research

Abstract

fetched live from OpenAlex

Medulloblastoma (MB) is the most common malignant pediatric brain tumor and consists of four subgroups: WNT, SHH, Group 3 and Group 4. Although drivers of Group 3 and 4 remain unknown, recurrent subchromosomal gains and losses frequently occurs. Current models to study MB include human cell lines, patient derived xenografts, and genetically engineered mouse models (GEMM). However, transcriptional profiles of human cell lines cluster separately from patient samples, and patient tumors are rare and difficult to culture. While GEMM reproducibly generate tumors resembling MB, modeling human patient-relevant chromosomal aberrations in mice would be difficult as mouse and human chromosomes do not align. To address these issues, we developed a human stem cell (hSC)-based model of MB. For proof of principle, we first generated iPSC from patients with Gorlin syndrome which is characterized by germline mutation in the SHH antagonist PTCH1 and predisposition to MB. The iPSC were differentiated to neuroepithelial stem (NES) cells (cell of origin of MB) and implanted orthotopically in mice. Tumors developed at long latency and histology resembled MB. Importantly, our model provides new functional insight of genes mutated in patient samples. For instance, CRISPR/Cas9 knockout of GSE1 (co-mutated with PTCH1 in SHH MB) in Gorlin NES cells accelerated tumor growth in vivo. Furthermore, MYCN, whose amplification occurs in SHH and Group 4, is sufficient to transform normal human NES cells. While GEMM of MYCN-driven MB align to Group 3, our hSC-based model more appropriately clustered to SHH. Using CRISPR/Cas9, we generated Group 4-relevant chromosomal deletions in human NES cells to evaluate its role in MB tumorigenesis. Thus, our hSC-based model of MB represents human patient tumors better than GEMM and can routinely evaluate candidate drivers of MB, including subchromosomal abnormalities.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.006
Threshold uncertainty score0.020

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0010.001
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0010.000
Research integrity0.0010.002
Insufficient payload (model declined to judge)0.0060.002

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.055
GPT teacher head0.331
Teacher spread0.276 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2017
Admission routes1
Has abstractyes

Explore more

Same venueNeuro-OncologySame topicCancer Cells and MetastasisFrench-language works237,207