MétaCan
Menu
Back to cohort
Record W2776432655 · doi:10.1371/journal.pgen.1007142

Integrated rare variant-based risk gene prioritization in disease case-control sequencing studies

2017· article· en· W2776432655 on OpenAlexfundno aff
Jhih‐Rong Lin, Quanwei Zhang, Ying Cai, Bernice E. Morrow, Zhengdong D. Zhang

Bibliographic record

VenuePLoS Genetics · 2017
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicCongenital heart defects research
Canadian institutionsnot available
FundersEunice Kennedy Shriver National Institute of Child Health and Human DevelopmentNational Institute of Child Health and Human DevelopmentSchool of Medicine, Emory UniversityNational Institutes of HealthNational Institute on AgingUniversity of TorontoEmory UniversityNational Human Genome Research InstituteCardiff UniversityNational Institute of General Medical SciencesNational Institute of Mental HealthAmerican Heart Association
KeywordsBiologyGenome-wide association studyGeneticsGeneGenetic associationComputational biologyDiseaseHuman genomePhenotypePrioritizationDNA sequencingGenomeSingle-nucleotide polymorphismGenotypeMedicine

Abstract

fetched live from OpenAlex

Rare variants of major effect play an important role in human complex diseases and can be discovered by sequencing-based genome-wide association studies.Here, we introduce an integrated approach that combines the rare variant association test with gene network and phenotype information to identify risk genes implicated by rare variants for human complex diseases.Our data integration method follows a 'discovery-driven' strategy without relying on prior knowledge about the disease and thus maintains the unbiased character of genome-wide association studies.Simulations reveal that our method can outperform a widely-used rare variant association test method by 2 to 3 times.In a case study of a small disease cohort, we uncovered putative risk genes and the corresponding rare variants that may act as genetic modifiers of congenital heart disease in 22q11.2deletion syndrome patients.These variants were missed by a conventional approach that relied on the rare variant association test alone. Author summaryCase-control sequencing studies are a promising design to uncover risk genes of human complex diseases implicated by rare variants.The recent development of different types of rare variant association tests has improved the statistical power to identify disease genes that harbor risk rare variants.However, none of the recent sequencing-based genomewide association studies identified robust disease association of rare variants or genes based on them.Due to limited sample sizes that can be feasibly achieved in real applications, current rare variant association tests can only generate marginal association signals for most risk genes.Here we proposed an integrated method that combined association signals with orthogonal biological evidence to uncover risk genes in sequencing studies.Designed to address the lack-of-power issue, our method was shown to effectively uncover risk genes with marginal association signals in data simulation.Indeed, in a real application demonstrated in our case study our method disclosed important risk genes of congenital heart disease in 22q11.2deletion syndrome that were missed by the previous study.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.024
metaresearch head score (Gemma)0.046
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Simulation or modeling · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: none
Teacher disagreement score0.024
Threshold uncertainty score0.127

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0240.046
Meta-epidemiology (narrow)0.0010.001
Meta-epidemiology (broad)0.0020.002
Bibliometrics0.0030.003
Science and technology studies0.0010.001
Scholarly communication0.0020.001
Open science0.0020.002
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.043
GPT teacher head0.316
Teacher spread0.273 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designSimulation or modeling
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations7
Published2017
Admission routes1
Has abstractyes

Explore more

Same venuePLoS GeneticsSame topicCongenital heart defects researchFrench-language works237,207