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Record W2789905222 · doi:10.1093/jcag/gwy008.264

A263 IBS-D MICROBIOTA INDUCES GUT-BRAIN DYSFUNCTION BY DISRUPTING INTESTINAL NEURAL AND IMMUNE PATHWAYS

2018· article· en· W2789905222 on OpenAlexaff
Viktoria Serkis, Giada De Palma, Sila Cocciolillo, Marc Pigrau, Jun Lü, Elena F. Verdú, Přemysl Berčík, Stephen M. Collins

Bibliographic record

VenueJournal of the Canadian Association of Gastroenterology · 2018
Typearticle
Languageen
FieldMedicine
TopicGastrointestinal motility and disorders
Canadian institutionsMcMaster University
Fundersnot available
KeywordsLamina propriaImmune systemImmunologyIrritable bowel syndromeBiologyGut floraPopulationMedicineInternal medicineEpithelium

Abstract

fetched live from OpenAlex

Irritable Bowel Syndrome (IBS) is a disorder of the gut-brain axis, with altered gut function and frequent psychiatric co-morbidity. We have previously shown in gnotobiotic mice that fecal microbiota from patients with IBS with diarrhea (IBS-D) and co-morbid anxiety induces faster gastrointestinal transit, gut barrier dysfunction, immune activation and anxiety-like behavior. However, the exact mechanisms underlying these abnormalities are not understood. To investigate and characterize the role of neural and immune factors in the microbiota-mediated alteration of gut physiology and behaviour. The expression of 72 murine genes related to neural, immune, and epithelial function was measured with NanoString nCounter® gene assay on total RNA extracted from colonic sections of mice colonized with microbiota from IBS-D patients or healthy volunteers. Colonic histology sections were stained for F4/80+ cells and multi-parameter flow cytometry was used to survey population of conventional and innate-like lymphocytes from various lymphoid compartments including the spleen, mesenteric lymph nodes, lamina propria, and intra-epithelial lymphocytes. Mice colonized with IBS-D microbiota had a strong upregulation of neural genes involved in secretomotor function (VIP, ChAT, CALB), visceral sensitivity (NR2D and GABA-B), innate immunity (CD11c, CCR2, GATA-3, and GPR44) and regulation of epithelial integrity and control of commensal microbiota (Trefoil factor 3 and Lysozyme) compared to mice colonized with healthy microbiota. Lamina propria macrophages levels were higher in IBS-D colonized mice compared to healthy controls. Multi-parameter flow cytometry revealed similar frequencies of conventional T cells (CD3+CD4+ and CD3+CD8+) or B cells (CD19+B220+) in the spleen, MLN, and intestinal compartments between mice with IBS-D microbiota vs. healthy controls. However, there was a higher relative frequency of TCRαβ-TCRγδ -RORγt+ cells in the colonic lamina propria of mice with IBS-D microbiota compared to healthy controls. Our results demonstrate that the intestinal microbiota from patients with IBS-D and co-morbid anxiety alters multiple immune and neural system pathways involved in the regulation of gut function. IBS-D microbiota appears to affect the innate but not the adaptive immune system, with macrophages and innate lymphoid cells playing a key role. CIHROntario Graduate Scholarship (OGS) - Masters

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.011

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0030.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.010
GPT teacher head0.221
Teacher spread0.211 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2018
Admission routes1
Has abstractyes

Explore more

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