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Record W2790138862 · doi:10.1093/jcag/gwy008.014

A13 PROTEOLYTIC BACTERIA PROMOTE INNATE IMMUNE ACTIVATION AND GLUTEN-INDUCED PATHOLOGY IN MICE.

2018· article· en· W2790138862 on OpenAlexaff
Heather J. Galipeau, A Caminero Fernandez, Justin L. McCarville, Suzanne M. Bernier, Joseph A. Murray, Jorge Blanco, Michael G. Surette, Elena F. Verdú

Bibliographic record

VenueJournal of the Canadian Association of Gastroenterology · 2018
Typearticle
Languageen
FieldMedicine
TopicCeliac Disease Research and Management
Canadian institutionsMcMaster UniversityPopulation Health Research Institute
Fundersnot available
KeywordsIntraepithelial lymphocyteGlutenEnteropathyImmune systemBiologyImmunologyMicrobiologyInnate immune systemNodMedicineDiseasePathologyGeneBiochemistry

Abstract

fetched live from OpenAlex

Celiac disease (CeD) is an immune-mediated enteropathy triggered by gluten in genetically susceptible individuals expressing the HLA-DQ2 or DQ8 genes. Only 2–4% of genetically susceptible individuals develop CeD, suggesting additional environmental triggers contribute to disease pathogenesis. Duodenal dysbiosis has been described in CeD, but no causative mechanisms been described. We have previously isolated Pseudomonas aeruginosa (Psa), an elastase-producing opportunistic pathogen, from the duodenum of active CeD patients that increases gluten peptide immunogenicity through its gluten metabolism. Here we determined whether microbial-derived elastolytic activity induces innate immune activation, a key step for enteropathy development in CeD. Altered-Schaedler flora (ASF)-colonized C57BL/6 (non-susceptible) and NOD/DQ8 (genetically susceptible) mice were supplemented with Psa prior to gliadin sensitization and challenge. Non-sensitized mice and mice supplemented with a Psa mutant lacking elastase activity (LasB) were used as controls. Small intestinal proteolytic activity, intraepithelial lymphocyte (IEL) counts and villus-to-crypt (V/C) ratios were measured following gluten challenge. ASF-colonized C57BL/6 and NOD/DQ8 mice had low proteolytic activity in small intestinal washes and were protected from gluten-induced pathology following gluten sensitization and challenge. In ASF-colonized mice supplemented with Psa (ASF-Psa), gluten exposure increased small intestinal Psa load, which paralleled increased elastase activity in small intestinal washes. ASF-Psa colonization led to increased small intestinal IELs, independent of gluten exposure, in both the non-susceptible C57BL/6 and the genetically susceptible NOD/DQ8 mice. However, reduction in small intestinal V/C ratios was only observed in ASF-Psa-colonized NOD/DQ8 mice exposed to gluten. No increase in IELs or enteropathy were observed in ASF mice supplemented with the LasB mutant. These results suggest that bacterial-derived elastase from opportunistic pathogens, such as P. aeruginosa, may directly promote IEL proliferation contributing to gluten-induced pathology in genetically susceptible hosts. Microbial-derived elastolytic activity constitutes a mechanism, in addition to its gluten metabolic activity, through which tolerance to this common dietary protein may be broken. It also opens the road to adjuvant therapies to the gluten-free diet based on protease inhibitory therapy. CIHR

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.008

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.001
Meta-epidemiology (broad)0.0000.001
Bibliometrics0.0010.000
Science and technology studies0.0000.001
Scholarly communication0.0010.001
Open science0.0000.000
Research integrity0.0010.002
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.010
GPT teacher head0.249
Teacher spread0.239 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2018
Admission routes1
Has abstractyes

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