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Record W2791797459 · doi:10.1093/jcag/gwy008.015

A14 INFLAMMASOME ACTIVATION COORDINATES INTESTINAL MUCOSAL DEFENSE AGAINST THE ENTERIC BACTERIAL PATHOGEN SALMONELLA ENTERICASEROVAR TYPHIMURIUM.

2018· article· en· W2791797459 on OpenAlexaff
Shauna M. Crowley, Franziska A. Graef, Joannie M. Allaire, Leigh A. Knodler, Bruce A. Vallance

Bibliographic record

VenueJournal of the Canadian Association of Gastroenterology · 2018
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicGut microbiota and health
Canadian institutionsBC Children's Hospital
Fundersnot available
KeywordsSalmonella entericaInflammasomeMicrobiologySalmonellaPathogenImmune systemIntestinal epitheliumBiologySecretionIntestinal mucosaInnate immune systemImmunologyInflammationBacteriaEpitheliumMedicineBiochemistry

Abstract

fetched live from OpenAlex

Intestinal epithelial cells (IECs) play a central role in the coordination of intestinal homeostasis. They must strike a careful balance to temper pro-inflammatory responses against the intestinal microbiota, while remaining vigilant and rapidly responsive when exposed to a noxious stimulus such as an enteric pathogen. One early response mechanism by which IECs engage in immune defense is through the activation of an IEC-specific inflammasome. This activation triggers the extrusion of infected IEC into the gut lumen, ultimately restricting Salmonella enterica serovar Typhimurium from escaping the gut and spreading systemically. However, it appears the role of the inflammasome in gut defense is not just restricted to IEC shedding but it also plays a key role in mucin secretion and antimicrobial lectin production. Here, we investigate the role of the inflammasome in mucosal defense against S. Typhimurium SL1344. This study employed a streptomycin pretreatment S. Typhimurium mouse infection model as well as various cell and immunofluorescent staining to characterize the role of the intestinal inflammasome. Streptomycin-pretreated C57BL/6, Casp1/11 deficient (−/−) and Casp11−/− mice were orally infected and S. Typhimurium loads determined for each respective tissue at 18h and 72h post infection. Increased pathogen burdens were observed for both caspase-deficient mice compared to wild type, with increased systemic spread seen in the mesenteric lymph nodes, liver and spleen at both time points for Casp1/11−/− mice but only at 72h for Casp11 −/−. Interestingly, at 18h, despite increased bacterial loads, cecal pathology scores were decreased in both Casp1/11−/− and Casp11−/− mice, as compared to wild type. This was accompanied by increased intracellular S. Typhimurium immunofluorescence staining. Also, cecal mucin layer thickness, as measured by Alcian Blue as well as MUC2 immunofluorescence staining were similar amongst all mouse strains before infection, but were decreased in both Casp1/11−/− and Casp11−/− mice as compared to wild type after infection. Finally, expression of the antimicrobial lectins REG3γ and REG3β were significantly decreased in Casp11−/− mice as compared to wild type mice and minimal staining for REG3β was observed in both caspase deficient mice. These results indicate that the gut epithelium utilizes inflammasome signaling to coordinate multiple layers of innate defense at the gut mucosal surface to ultimately restrict enteric pathogen infections and systemic spread. CCC, CIHRNSERC, UBC

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.005

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.006
GPT teacher head0.214
Teacher spread0.208 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2018
Admission routes1
Has abstractyes

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