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Record W2792754109 · doi:10.1093/jcag/gwy009.087

A87 MACROPHAGE-ACTIVATED INFLAMMASOME MEDIATES RECOVERY OF INTESTINAL EPITHELIAL CELLS DURING INFECTION

2018· article· en· W2792754109 on OpenAlexaff
Michael Bording‐Jorgensen, Heather Armstrong, E Zwaigenbaum, Eytan Wine

Bibliographic record

VenueJournal of the Canadian Association of Gastroenterology · 2018
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicGut microbiota and health
Canadian institutionsUniversity of Alberta
Fundersnot available
KeywordsCitrobacter rodentiumInflammasomeTight junctionOccludinMicrobiologyMacrophageBiologyCell biologyInflammationClaudinBarrier functionChemistryImmunologyIn vitroPathogenBiochemistry

Abstract

fetched live from OpenAlex

Nod-like receptor protein-3 (NLRP3) inflammasomes have been linked to various inflammatory conditions such as inflammatory bowel diseases (IBD). Conditions associated with the inflammasomes are typically characterized by an overabundance of interleukin (IL)-1β with the exception of IBD, where its dysregulation results in hypoproduction. The mouse pathogen Citrobacter rodentium, which causes IBD-like colitis, is used to understand the dynamic relationship between pathogens, the inflammasome and the epithelial barrier. We have recently shown that ATP-activated macrophages increase clearance of C. rodentium in vitro. Our hypothesis is that inflammasome activation will increase macrophage recruitment and facilitate protection of the epithelial barrier. The aim was to determine the role the inflammasome plays during macrophage-epithelial interaction during C. rodentium infection Mouse colonic epithelial cells (CMT-93) were seeded on the bottom of collagen coated transwells until tight junctions were formed using transepithelial electrical resistance (TEER). The CMT-93 cells were infected with C. rodentium on the apical membrane. ATP and LPS were used to activate the inflammasome in macrophages with YVAD as an inhibitor and added to the basolateral membrane, with tight junction permeability measured using TEER. Immunofluorescence was done using confocal microscopy looking at tight junction proteins ZO-1, occluding and claudin 1. Adherence of C. rodentium to the epithelial barrier was assessed using serial dilutions and plating on LB agar at 37°C overnight. ATP-activated macrophages were increased at the apical membrane during C. rodentium infection of epithelial cells. Tight junction proteins Claudin-1, Occludin, and ZO-1 were found on macrophages using immunofluorescence. The presence of epithelial ZO-1 rings were observed during C. rodenitum infection through immunofluorescence. C. rodentium adherence was found to be significantly reduced, together with an increase in epithelial TEER recovery; this was inhibited using YVAD. These data suggest that the inflammasome is an important mediator for pathogen clearance by macrophages. Some individuals with Crohn Disease that have a decreased ability to produce IL-1β may be more susceptible to infection, as they cannot clear a pathogen. Furthermore, the role of the epithelial cell in this relationship is critical to understanding why the epithelial barrier in IBD patients is disrupted. CCCCCA

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.011

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0030.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.005
GPT teacher head0.210
Teacher spread0.206 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2018
Admission routes1
Has abstractyes

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