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Record W2795137078 · doi:10.1152/ajpheart.00689.2017

Influence of myocardial oxygen demand on the coronary vascular response to arterial blood gas changes in humans

2018· article· en· W2795137078 on OpenAlexafffund
Tyler D. Vermeulen, Lindsey M. Boulet, Mike Stembridge, Alexandra M. Williams, James D. Anholm, Prajan Subedi, Chris Gasho, Philip N. Ainslie, Eric O. Feigl, Glen E. Foster

Bibliographic record

VenueAmerican Journal of Physiology-Heart and Circulatory Physiology · 2018
Typearticle
Languageen
FieldMedicine
TopicHeart Rate Variability and Autonomic Control
Canadian institutionsUniversity of British Columbia, Okanagan CampusUniversity of British Columbia
FundersNatural Sciences and Engineering Research Council of CanadaGovernment of Canada
KeywordsCardiologyHypoxia (environmental)EsmololMedicineInternal medicineBlood pressureHeart rateVasodilationAnesthesiaOxygenChemistry

Abstract

fetched live from OpenAlex

It remains unclear if the human coronary vasculature is inherently sensitive to changes in arterial Po 2 and Pco 2 or if coronary vascular responses are the result of concomitant increases in myocardial O 2 consumption/demand ([Formula: see text]). We hypothesized that the coronary vascular response to Po 2 and Pco 2 would be attenuated in healthy men when [Formula: see text] was attenuated with β 1 -adrenergic receptor blockade. Healthy men (age: 25 ± 1 yr, n = 11) received intravenous esmolol (β 1 -adrenergic receptor antagonist) or volume-matched saline in a double-blind, randomized crossover study and were exposed to poikilocapnic hypoxia, isocapnic hypoxia, and hypercapnic hypoxia. Measurements made at baseline and after 5 min of steady state at each gas manipulation included left anterior descending coronary blood velocity (LAD V ; Doppler echocardiography), heart rate, and arterial blood pressure. LAD V values at the end of each hypoxic condition were compared between esmolol and placebo. The rate-pressure product (RPP) and left ventricular mechanical energy (ME LV ) were calculated as indexes of [Formula: see text]. All gas manipulations augmented RPP, ME LV , and LAD V , but only RPP and ME LV were attenuated (4–18%) after β 1 -adrenergic receptor blockade ( P < 0.05). Despite attenuated RPP and ME LV responses, β 1 -adrenergic receptor blockade did not attenuate the mean LAD V vasodilatory response compared with placebo during poikilocapnic hypoxia (29.4 ± 2.2 vs. 27.3 ± 1.6 cm/s) and isocapnic hypoxia (29.5 ± 1.5 vs. 30.3 ± 2.2 cm/s). Hypercapnic hypoxia elicited a feedforward coronary dilation that was blocked by β 1 -adrenergic receptor blockade. These results indicate a direct influence of arterial Po 2 on coronary vascular regulation that is independent of [Formula: see text]. NEW & NOTEWORTHY In humans, arterial hypoxemia led to an increase in epicardial coronary artery blood velocity. β 1 -Adrenergic receptor blockade did not diminish the hypoxemic coronary response despite reduced myocardial O 2 demand. These data indicate hypoxemia can regulate coronary blood flow independent of myocardial O 2 consumption. A plateau in the mean left anterior descending coronary artery blood velocity-rate-pressure product relationship suggested β 1 -adrenergic receptor-mediated, feedforward epicardial coronary artery dilation. In addition, we observed a synergistic effect of Po 2 and Pco 2 during hypercapnic hypoxia.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.003

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.010
GPT teacher head0.245
Teacher spread0.235 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations8
Published2018
Admission routes2
Has abstractyes

Explore more

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