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Evolution of genomic instability in metastatic cancer.

2018· article· en· W2890257235 on OpenAlexaff
Eric Y. Stutheit-Zhao, Erin Pleasance, Martin Jones, Yaoqing Shen, Caralyn Reisle, Andrew J. Mungall, Richard A. Moore, Yongjun Zhao, Daniel J. Renouf, Janessa Laskin, Marco A. Marra, Steven J.M. Jones

Bibliographic record

VenueJournal of Clinical Oncology · 2018
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicCancer Genomics and Diagnostics
Canadian institutionsGenome British ColumbiaCanada's Michael Smith Genome Sciences CentreBC Cancer Agency
Fundersnot available
KeywordsMutationCancerGenome instabilityMetastasisGermline mutationMedicineDiseaseGeneticsBiologyComputational biologyGenePathologyDNA damage

Abstract

fetched live from OpenAlex

12008 Background: Although metastasis underlies up to 90% of cancer-related mortality, genomic instability and mutation signatures are mostly studied in primary tumours. Mutation signatures are patterns of somatic mutation resulting from specific mutational processes (i.e. tobacco/UV exposure) and often evolve over time. Recent studies suggest that certain mutation signatures may predict chemotherapy response. Understanding mutational processes in metastatic cancers could uncover actionable targets and refine the understanding of progression and drug resistance. Methods: As part of the BC Cancer Agency Personalized Oncogenomics Project, mutation signatures were deciphered from 571 metastatic whole genomes from 12 cancer types totalling 13,249,678 somatic mutations. We created a novel Bayesian hiearchical model named SignIT (github.com/eyzhao/SignIT) to track temporal evolution of mutation signatures. Using real and simulated data, we showed that SignIT decomposes signatures and their temporal evolution more accurately than comparable methods. Previous chemotherapy treatments were catalogued for all patients by retrospective review. Results: We discovered 21 distinct mutation signatures, including 9 novel signatures (numbered M1-M9). Mutational processes associated with aging and cigarette smoke were early-arising. Signature 17 and M2 were consistently late-arising across cancer types and metastatic sites. Prior treatment with platinum-based chemotherapy was associated with depression of the homologous recombination deficiency signature 3 (p = 0.03). Platinum exposure was also associated with late elevation of signature 17. Conclusions: To date, this is the largest study of metastatic cancer whole genomes. Our findings revealed 9 novel mutation signatures, including potential markers of late disease and metastasis. We also observed temporal evolution of mutation signatures correlated with chemotherapy exposures. The association of decreasing signature 3 activity with platinum exposure suggests the restoration of homologous recombination as a resistance mechanism. These findings highlight the complexity of metastatic cancers, and the variety of factors which impact their mutagenesis.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.003
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.009
Threshold uncertainty score0.017

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.003
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.001
Bibliometrics0.0010.001
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.073
GPT teacher head0.442
Teacher spread0.369 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2018
Admission routes1
Has abstractyes

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