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Record W2899550130

Transformation of normal NIH-3T3 cells into highly oncogenic and invasive cancer cells by MBD2 overexpression

2007· article· en· W2899550130 on OpenAlexaffabout
Stephen D. Andrews, Bushra Ateeq, Jérôme Torrisani, Ana C. D’Alessio, Alexander Unterberger, Jing-Ni Ou, Marilène Paquet, Shafaat A. Rabbani, Moshe Szyf

Bibliographic record

VenueCancer Research · 2007
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicEpigenetics and DNA Methylation
Canadian institutionsMcGill University
Fundersnot available
KeywordsDNA methylationBiologyCarcinogenesisCancer researchCell cultureCancer cellGene knockdownMalignant transformationMolecular biology3T3 cellsCell biologyCancerTransfectionGene expressionGeneGenetics
DOInot available

Abstract

fetched live from OpenAlex

3925 Introduction: Cancer cells are hallmarked by global DNA hypomethylation and regional hypermethylation of tumor suppressor genes. The latter has been extensively studied while not much attention has been given to global DNA hypomethylation. MBD2 is the only protein that has been shown to directly remove the methyl group from CG di-nucleotides; though, it has also been reported to be a transcriptional repressor. Inhibition of MBD2 attenuates tumorigenesis, metastasis, and reverses the hypomethylation of metastatic genes in cell culture and in vivo. Additionally, germ line deletion of the mbd2 gene protects mice from intestinal tumors. Therefore, we tested the hypothesis that MBD2 causes oncogenic and metastatic transformation of cells by triggering global hypomethylation. Results: MBD2 and a mutant MBD2 lacking the MBD domain were stably and transiently expressed in mouse fibroblast NIH-3T3 cells. We determined that MBD2 induces global hypomethylation and histone H3 lysine 9 acetylation in an MBD domain dependent manner. Several assays revealed that the MBD2 cell lines, but not the mutant nor the control cell lines, were transformed and highly invasive in cell culture. Only the MBD2 cell lines were highly tumorigenic and invasive when injected into Nude mice while also showing a remarkable ability to invade and degrade bone tissue when injected into the tibia of SCID mice. We also determined that MBD2 is required for maintaining a transformed state triggered by activated HA-RAS in NIH-3T3 cells. MBD2 knockdown in these cells attenuated transformation and increased DNA methylation supporting our hypothesis. To delineate the effects of MBD2 overexpression on specific gene expression, a genomic approach using affymetrix expression arrays was employed. Four hundred and forty four genes were identified to have their mRNA expression altered by MBD2 expression. Genes involved in tumorgenesis, bone degradation, or with no known function that were induced in both MBD2 and HA-Ras expressing cell lines were chosen for further study. Knocking down selected candidate genes9 mRNA in MBD2 overexpressing NIH-3T3 cells resulted in attenuated tumorigenesis while knocking down MBD2 in HA-RAS cells significantly reduced these genes9 expression levels. CHIP assay of the promoter and bisulfite sequencing of the same regions for the candidate genes revealed that MBD2 binds only to regions associated with demethylated DNA and that these regions are associated with H3 lysine 9 acetylation. Summary: Our results support the hypothesis that MBD2, through activation of global cancer pathways, transforms normal fibroblast cells into highly oncogenic, invasive, and metastatic cancer cells; therefore, MBD2 is a novel cancer therapeutic candidate. Supported by a grant from the National Cancer Institute of Canada to MS.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.007

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.001
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.024
GPT teacher head0.358
Teacher spread0.335 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2007
Admission routes2
Has abstractyes

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