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Record W2899686888 · doi:10.1093/neuonc/noy148.1021

STEM-14. GROWTH FACTOR RECEPTOR CO-INHERITANCE DURING ASYMMETRIC CELL DIVISION DRIVES THE CANCER STEM CELL PHENOTYPE

2018· article· en· W2899686888 on OpenAlexaff
Masahiro Hitomi, Anastasia Chumakova, Stephanie Jarvis, Neha Anand, Daniel J. Silver, Arnon Møldrup Knudsen, Bridget Corrigan, Peter S. Yoo, Upashruti Agrawal, Kayla E. Pfaff, Vid Yogeswaran, Malini Kaminen, Masahiko Sato, Sachiko Sato, Bjarne Winther Kristensen, Justin D. Lathia

Bibliographic record

VenueNeuro-Oncology · 2018
Typearticle
Languageen
FieldMedicine
TopicCancer Cells and Metastasis
Canadian institutionsUniversité Laval
Fundersnot available
KeywordsStem cellBiologyCancer stem cellCell biologyCell divisionMitosisAsymmetric cell divisionCancer researchCell fate determinationGrowth factorReceptorCellTranscription factorGeneticsGene

Abstract

fetched live from OpenAlex

An asymmetric cell division (ACD) produces a stem cell and a differentiating progeny. Thus ACD ensures the generation of organs with heterogeneous cell populations without depleting pools of stem cells with regenerative capacity. Cancer stem cells (CSCs), which are similar to normal stem cells, can self-renew and regenerate tumors with cellular heterogeneity. CSCs are resistant to therapy and play a critical role in tumor recurrence. ACD has been detected in CSCs from many types of tumors, but its role in CSC fate decision has yet to be fully elucidated. A remaining technical limitation is that ACD is often defined retrospectively based on the observation of an asymmetric fate choice by CSC progeny. We previously demonstrated asymmetric inheritance of a surrogate CSC marker, CD133, during mitosis. To prospectively analyze the biological role of this inheritance asymmetry, we have developed a GFP reporter system capable of monitoring the degree of asymmetry of the CSC marker during mitosis. This reporter also revealed the asymmetric co-inheritance of growth factor receptors, the activation of which overrode the effect of a differentiation-inducing condition that suppresses self-renewal capacity and therapeutic resistance of CSCs. Preliminary time lapse-based lineage tracing detected that daughter cells that inherited higher levels of growth factor receptors based on GFP-reporter signal intensity express higher levels of a core stem cell transcription factor compared to their sister cells that inherited lower levels of growth factor receptors. These data suggest that asymmetrically co-inherited growth factor receptors promote the stem cell state in one of the progeny of a CSC undergoing ACD, ensuring the persistence of a therapeutically resistant population.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.004
Threshold uncertainty score0.013

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0040.002

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.026
GPT teacher head0.295
Teacher spread0.269 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2018
Admission routes1
Has abstractyes

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